E
Eduardo Molina-Jijón
Researcher at Instituto Politécnico Nacional
Publications - 32
Citations - 1921
Eduardo Molina-Jijón is an academic researcher from Instituto Politécnico Nacional. The author has contributed to research in topics: Oxidative stress & Curcumin. The author has an hindex of 21, co-authored 29 publications receiving 1603 citations. Previous affiliations of Eduardo Molina-Jijón include National Autonomous University of Mexico.
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Journal ArticleDOI
Renoprotective effect of the antioxidant curcumin: Recent findings
Joyce Trujillo,Yolanda I. Chirino,Eduardo Molina-Jijón,Ana Cristina Andérica-Romero,Edilia Tapia,José Pedraza-Chaverri +5 more
TL;DR: The information presented in this paper identifies curcumin as a promising renoprotective molecule against renal injury.
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Curcumin prevents Cr(VI)-induced renal oxidant damage by a mitochondrial pathway
Eduardo Molina-Jijón,Edilia Tapia,Cecilia Zazueta,Mohammed El Hafidi,Zyanya Lucía Zatarain-Barrón,Rogelio Hernández-Pando,Omar Noel Medina-Campos,Guillermo Zarco-Márquez,Ismael Torres,José Pedraza-Chaverri +9 more
TL;DR: It was found that this antioxidant accumulated in kidney and activated Nrf2 at the time when K( 2)Cr(2)O(7) was injected, suggesting that both direct and indirect antioxidant effects are involved in the protective effects of curcumin.
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Curcumin induces Nrf2 nuclear translocation and prevents glomerular hypertension, hyperfiltration, oxidant stress, and the decrease in antioxidant enzymes in 5/6 nephrectomized rats.
Edilia Tapia,Virgilia Soto,Karla M. Ortiz-Vega,Guillermo Zarco-Márquez,Eduardo Molina-Jijón,Magdalena Cristóbal-García,José Santamaría,Wylly Ramsés García-Niño,Francisco Correa,Cecilia Zazueta,José Pedraza-Chaverri +10 more
TL;DR: It is concluded that the protective effect of curcumin against 5/6NX-induced glomerular and systemic hypertension, hyperfiltration, renal dysfunction, and renal injury was associated with the nuclear translocation of Nrf2 and the prevention of both oxidant stress and the decrease of antioxidant enzymes.
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Titanium dioxide nanoparticles impair lung mitochondrial function.
Verónica Freyre-Fonseca,Norma L. Delgado-Buenrostro,Emma Berta Gutiérrez-Cirlos,Claudia Marissa Calderón-Torres,Tecilli Cabellos-Avelar,Yesennia Sánchez-Pérez,Enrique Pinzón,Ismael Torres,Eduardo Molina-Jijón,Cecilia Zazueta,José Pedraza-Chaverri,Claudia M. García-Cuellar,Yolanda I. Chirino +12 more
TL;DR: TiO(2) NPs decreases NADH levels and impairs ΔΨ(m) and mitochondrial function accompanied by ROS generation during mitochondrial respiration and this effect is associated with mitochondrial dysfunction and redox unbalance.
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Protective effect of sulforaphane pretreatment against cisplatin-induced liver and mitochondrial oxidant damage in rats.
Leobardo Gaona-Gaona,Eduardo Molina-Jijón,Edilia Tapia,Cecilia Zazueta,Rogelio Hernández-Pando,Mariel Calderón-Oliver,Guillermo Zarco-Márquez,Enrique Pinzón,José Pedraza-Chaverri +8 more
TL;DR: It was found that SFN prevents CIS-induced hepatic damage, oxidant stress and decreased activity of antioxidant enzymes in liver and mitochondria and mitochondrial alterations in oxygen consumption and decreases activity of mitochondrial complex I.