E
Ellinor Kenne
Researcher at Karolinska Institutet
Publications - 31
Citations - 1762
Ellinor Kenne is an academic researcher from Karolinska Institutet. The author has contributed to research in topics: Inflammation & Factor XII. The author has an hindex of 19, co-authored 28 publications receiving 1406 citations. Previous affiliations of Ellinor Kenne include Syracuse University & Karolinska University Hospital.
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Journal ArticleDOI
Contact system revisited: an interface between inflammation, coagulation, and innate immunity.
TL;DR: This review summarizes how the contact system acts as the cross‐road of inflammation, coagulation, and innate immunity.
Journal ArticleDOI
Neutrophil primary granule proteins HBP and HNP1-3 boost bacterial phagocytosis by human and murine macrophages.
Oliver Soehnlein,Ylva Kai-Larsen,Robert Frithiof,Ole E. Sørensen,Ellinor Kenne,Karin Scharffetter-Kochanek,Einar E. Eriksson,Heiko Herwald,Birgitta Agerberth,Lennart Lindbom +9 more
TL;DR: What may be a novel role for PMN granule proteins in regulating the immune response to bacterial infections is attributes to heparin-binding protein and human neutrophil peptides 1-3 (HNP1-3) as the mediators of the macrophage response to PMN secretion.
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Neutrophil depletion reduces edema formation and tissue loss following traumatic brain injury in mice
TL;DR: The results suggest that neutrophils are involved in the edema formation, but not the extravasation of large proteins, as well as contributing to cell death and tissue loss following TBI in mice.
Journal ArticleDOI
Distinct Infiltration of Neutrophils in Lesion Shoulders in ApoE−/− Mice
Pierre Rotzius,Sebastian Thams,Oliver Soehnlein,Oliver Soehnlein,Ellinor Kenne,Chi-Nan Tseng,Niklas K. Björkström,Karl-Johan Malmberg,Lennart Lindbom,Einar E. Eriksson +9 more
TL;DR: In this paper, neutrophils made up for 1.8% of CD45+ leukocytes in the aortic wall of ApoE−/−/LysEGFP/EGFP mice and their contribution relative to monocyte/macrophages within lesions is approximately 1:3.
Journal ArticleDOI
Polyphosphate nanoparticles on the platelet surface trigger contact system activation.
Johan J F Verhoef,Arjan D. Barendrecht,Katrin F. Nickel,Katrin F. Nickel,Kim Dijkxhoorn,Ellinor Kenne,Linda Labberton,Owen J. T. McCarty,Raymond M. Schiffelers,Harry F. G. Heijnen,Antoni P. A. Hendrickx,Huub Schellekens,Marcel H.A.M. Fens,Steven de Maat,Thomas Renné,Thomas Renné,Coen Maas +16 more
TL;DR: In this article, the authors identify membrane-associated polyphosphate in a nanoparticle state on the surface of activated platelets and mechanistically link the procoagulant activity of platelets with the activation of coagulation factor XII.