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Elliott M. Antman

Researcher at Brigham and Women's Hospital

Publications -  738
Citations -  187175

Elliott M. Antman is an academic researcher from Brigham and Women's Hospital. The author has contributed to research in topics: Myocardial infarction & TIMI. The author has an hindex of 161, co-authored 716 publications receiving 179462 citations. Previous affiliations of Elliott M. Antman include Duke University & Katholieke Universiteit Leuven.

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Isolated Right Ventricular Infarction

TL;DR: A 47-year-old man with no history of cardiac disease presented to a hospital, reporting severe substernal chest pressure associated with bilateral arm weakness, and was treated with fibrinolytic therapy and transferred to another hospital for catheterization.
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Evidence-Based Coronary Care

TL;DR: Patients enjoyed the benefit of a halving of the in-hospital mortality rate for acute myocardial infarction but remained susceptible to the late consequences of large infarctions: heart failure and malignant ventricular arrhythmias.
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Methodologic and clinical validation of the TIMI myocardial perfusion grade in acute myocardial infarction.

TL;DR: DSA validates that TMPG grade 3 is associated with normal kinetics of myocardial perfusion, and this likely accounts for the low 30 day mortality observed among those patients with TFG 3 and TMPG 3.
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Differential effects of reperfusion on incidence of ventricular arrhythmias and recovery of ventricular function at 4 days following coronary occlusion

TL;DR: At 4 days post occlusion in a dog model, spontaneous ventricular arrhythmias are reduced by reperfusion within 4 hours, while return of ventricular function is only improved by reperFusion within approximately 1 hour of coronary occlusions.
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Association of a History of Systemic Hypertension With Mortality, Thrombotic, and Bleeding Complications Following Non-ST-Segment Elevation Acute Coronary Syndrome

TL;DR: It was concluded that chronic hypertension remains an independent marker for major short‐ and long‐term cardiac adverse outcomes after non‐ST‐segment elevation acute coronary syndrome.