scispace - formally typeset
E

Elliott M. Antman

Researcher at Brigham and Women's Hospital

Publications -  738
Citations -  187175

Elliott M. Antman is an academic researcher from Brigham and Women's Hospital. The author has contributed to research in topics: Myocardial infarction & TIMI. The author has an hindex of 161, co-authored 716 publications receiving 179462 citations. Previous affiliations of Elliott M. Antman include Duke University & Katholieke Universiteit Leuven.

Papers
More filters
Journal ArticleDOI

Population and Personalized Medicine in the Modern Era

TL;DR: Clinical research has reached a pivotal moment, not only with the exponential expansion of tools for data capture as well as data sources, but also with the opportunity to reevaluate how to integrate the information to optimize medical decision making.
Journal ArticleDOI

Association of epicardial and tissue-level reperfusion with left ventricular end-diastolic pressures in ST-elevation myocardial infarction.

TL;DR: Higher post-fibrinolytic LVEDP was associated with age ≥65, female gender, Killip Class II–IV on presentation, and LAD culprit location, and elevated LVEDp was independently associated with a greater incidence of in-hospital and 30-day congestive heart failure.
Journal ArticleDOI

Association of non-steroidal anti-inflammatory drugs with outcomes in patients with ST-segment elevation myocardial infarction treated with fibrinolytic therapy: an ExTRACT-TIMI 25 analysis

TL;DR: Among STEMI patients treated with a fibrinolytic agent and aspirin, use of NSAIDs in the week preceding the incident event was associated with a higher incidence of MI, the composite of death and MI as well as the compositeof death, MI, severe heart failure and shock at 30 days.
Journal ArticleDOI

Degree of residual stenosis in the culprit coronary artery after thrombolytic administration (Thrombolysis in Myocardial Infarction [TIMI] Trials)

TL;DR: Among patients with patent arteries, a residual stenosis of <50% was associated with a significantly lower composite of in-hospital death, myocardial infarction, and congestive heart failure and these patients have less complex lesions with greater thrombus burdens and better clinical outcomes.