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Esper G. Kallas

Researcher at University of São Paulo

Publications -  291
Citations -  15157

Esper G. Kallas is an academic researcher from University of São Paulo. The author has contributed to research in topics: Population & Medicine. The author has an hindex of 51, co-authored 255 publications receiving 12763 citations. Previous affiliations of Esper G. Kallas include University of Rochester & Global Virus Network.

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HIV-1-Infected Children on HAART: Immunologic Features of Three Different Levels of Viral Suppression

TL;DR: Investigation of how highly active anti‐retroviral therapy (ART) alter blood lymphocyte subpopulations in HIV‐1‐infected children found good, partial and poor responses to ART on the basis of viral load measurement in blood.
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Cytomegalovirus Antigenemia in Acquired Immunodeficiency Syndrome Patients With Untreated Cytomegalovirus Retinitis

TL;DR: The CMV antigenemia assay may be a simple and rapid means of identifying those patients with unilateral retinitis at highest risk of developing CMV Retinitis of the fellow eye or of visceral CMV disease if intravitreal injections or implants are used as sole treatment for CMVretinitis.
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Subsets of memory CD4+ T cell and bactericidal antibody response to Neisseria meningitidis serogroup C after immunization of HIV-infected children and adolescents.

TL;DR: Investigation of associations between bactericidal antibody response induced by MenC vaccine and the frequency and activation profile (expression of CD38, HLA-DR and CCR5 molecules) of total CD4+ memory T cell sub-populations in HIV-1-infected children and adolescents indicates that less differentiated CD+ T cells, like TCM may be constantly differentiating into intermediate and later differentiated CD4- T cell subsets.
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Prevalence, Incidence Density, and Genotype Distribution of GB Virus C Infection in a Cohort of Recently HIV-1-Infected Subjects in Sao Paulo, Brazil

TL;DR: This study provides the first report of the GBV-C prevalence at the time of diagnosis of HIV-1 and the incidence density of GBv-C infection in one year and the predominant genotype is 2b.