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Reinaldo Salomão

Researcher at Federal University of São Paulo

Publications -  192
Citations -  6303

Reinaldo Salomão is an academic researcher from Federal University of São Paulo. The author has contributed to research in topics: Sepsis & Intensive care. The author has an hindex of 36, co-authored 180 publications receiving 5445 citations. Previous affiliations of Reinaldo Salomão include University of São Paulo.

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Device-associated nosocomial infections in 55 intensive care units of 8 developing countries

TL;DR: Device-associated infections, particularly ventilator-associated pneumonia, central venous catheterrelated bloodstream infections, and catheter-associated urinary tract infections pose the greatest threat to patient safety in the ICU, according to the initial findings of an International Nosocomial Infection Control Consortium (INICC) surveillance study.
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International Nosocomial Infection Control Consortiu (INICC) report, data summary of 43 countries for 2007-2012. Device-associated module

Victor D. Rosenthal, +825 more
TL;DR: The results of an International Nosocomial Infection Control Consortium (INICC) surveillance study from January 2007-December 2012 in 503 intensive care units (ICUs) in Latin America, Asia, Africa, and Europe were reported in this paper.
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High rate of non-albicans candidemia in Brazilian tertiary care hospitals.

TL;DR: Nosocomial candidemias in tertiary hospitals are caused predominantly by non-albicans species, which are rarely fluconazole resistant, and could not be related to the previous use of azoles.
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The epidemiology of sepsis in Brazilian intensive care units (the Sepsis PREvalence Assessment Database, SPREAD): an observational study

Flávia Ribeiro Machado, +307 more
TL;DR: The results show the burden of sepsis in resource-limited settings, highlighting the need to establish programmes aiming for sepsi prevention, early diagnosis, and adequate treatment.
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Bacterial sensing, cell signaling, and modulation of the immune response during sepsis.

TL;DR: This review discusses aspects of bacterial recognition and induced cellular activation during sepsis, and focuses on LPS and host interactions as a clue to understand microorganisms sensing and cell signaling.