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F.G. de Mello

Researcher at Federal University of Rio de Janeiro

Publications -  26
Citations -  1120

F.G. de Mello is an academic researcher from Federal University of Rio de Janeiro. The author has contributed to research in topics: Kainate receptor & GABAergic. The author has an hindex of 17, co-authored 26 publications receiving 1080 citations. Previous affiliations of F.G. de Mello include Northwestern University.

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Uptake of apoptotic cells drives the growth of a pathogenic trypanosome in macrophages.

TL;DR: The results suggest that continual lymphocyte apoptosis and phagocytosis of apoptotic cells by macrophages have a role in parasite persistence in the host, and that cyclooxygenase inhibitors have potential therapeutic application in the control of parasite replication and spread in Chagas' disease.
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Regulation of dopamine- and adenosine-dependent adenylate cyclase systems of chicken embryo retina cells in culture.

TL;DR: Evidence is obtained that receptor-stimulated adenylate cyclase activity [ATP pyrophosphate-lyase (cyclizing), EC 4.6.1] is regulated physiologically in both embryonic and mature neurons, and that regulation of the dopamine receptor system begins early in development and continues to function in mature synapses.
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Trophic activity derived from bone marrow mononuclear cells increases peripheral nerve regeneration by acting on both neuronal and glial cell populations.

TL;DR: A rat model of complete sciatic nerve transection was used to evaluate the effect of bone marrow mononuclear cells (BMMC) transplanted to the injury site immediately after lesion as mentioned in this paper.
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Expression of A1 adenosine receptors modulating dopamine-dependent cyclic AMP accumulation in the chick embryo retina.

TL;DR: The data suggest the existence of two populations of D1 dopamine receptors in the chick embryo retina—one that is negatively modulated by adenosine and another that is not under purinergic control.
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Aspartate as a selective NMDA receptor agonist in cultured cells from the avian retina.

TL;DR: The results indicate that in the retina, l-aspartate modulates its excitatory function on a set of GABAergic cells via the selective activation of NMDA receptors.