F. X. Bosch

TL;DR: There is a very strong association between HPV 16, 18, 31, 33 and 35 and invasive cervical cancer and that this association is probably causal.

TL;DR: A population‐based case‐control study of cervical cancer was conducted in Spain and Colombia to assess the relationship between cervical cancer and exposure to human papillomavirus (HPV), selected aspects of sexual and reproductive behaviour, use of oral contraceptives, screening practices and smoking.

TL;DR: Number of sexual partners, early age at first intercourse, parity and duration of oral contraceptive use were significantly associated with an increased risk of cervical cancer and the very high risks found in this study further implicate this virus in the aetiology of cervicalcancer.

TL;DR: Of the non‐dietary factors, sedentariness in the workplace and urban residence were the only risk factors identified, and consumption of vegetables was highly protective, irrespective of the cooking procedures.

TL;DR: These findings could be interpreted as indicating that past infections with HSV‐2, N. gonorrhoeae and C. trachomatis are surrogate markers of HPV, but because HPV DNA may have escaped detection, it cannot exclude that these STAs are also of separate etiologic significance.