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Gualtiero Pelosi

Researcher at National Research Council

Publications -  125
Citations -  1729

Gualtiero Pelosi is an academic researcher from National Research Council. The author has contributed to research in topics: Coronary artery disease & Medicine. The author has an hindex of 20, co-authored 117 publications receiving 1512 citations. Previous affiliations of Gualtiero Pelosi include University of Pisa.

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In vivo quantitative ultrasonic evaluation of myocardial fibrosis in humans.

TL;DR: In vivo on-line quantitative ultrasound analysis is feasible in man and reliably identifies variations in the regional extent of fibrosis in human myocardium as estimated by quantitative histology of endomyocardial biopsies.
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Myocardial Blood Flow Response to Pacing Tachycardia and to Dipyridamole Infusion in Patients With Dilated Cardiomyopathy Without Overt Heart Failure A Quantitative Assessment by Positron Emission Tomography

TL;DR: In patients with DCM without overt heart failure, the abnormalities in vasodilating capability can be present despite normal hemodynamics; progression of the disease is associated with more depressed myocardial perfusion.
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In vivo radiofrequency-based ultrasonic tissue characterization of the atherosclerotic plaque.

TL;DR: Quantitative analysis of integrated backscatter of the arterial wall is feasible in humans and provides an operator-independent assessment of plaque echoic structure, and integrated back scatter is effective in distinguishing lipidic, fibrotic, and calcific components in human atherosclerotic plaques.
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Impaired sympathetic response before intradialytic hypotension: a study based on spectral analysis of heart rate and pressure variability

TL;DR: It is concluded that standard haemodialysis activates a marked and reversible sympathetic response in both stable and unstable uraemic patients, however, in unstable patients, such activation is impaired in late dialysis, therefore contributing to the onset of the hypotensive crisis.
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Signal-averaged ECG abnormalities in haemodialysis patients. Role of dialysis.

TL;DR: LP were detected in a significant proportion of dialysis patients, probably related to underlying CAD with left ventricular dysfunction, and could be explained by the acute reduction in serum potassium levels.