F
Fanny E. Hartmann
Researcher at Université Paris-Saclay
Publications - 38
Citations - 1561
Fanny E. Hartmann is an academic researcher from Université Paris-Saclay. The author has contributed to research in topics: Genome & Population. The author has an hindex of 15, co-authored 36 publications receiving 1000 citations. Previous affiliations of Fanny E. Hartmann include ETH Zurich & University of Paris.
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Journal ArticleDOI
A small secreted protein in Zymoseptoria tritici is responsible for avirulence on wheat cultivars carrying the Stb6 resistance gene.
Ziming Zhong,Thierry C. Marcel,Fanny E. Hartmann,Xin Ma,Clémence Plissonneau,Clémence Plissonneau,Marcello Zala,Aurélie Ducasse,Johann Confais,Jérome Compain,Nicolas Lapalu,Joelle Amselem,Bruce A. McDonald,Daniel Croll,Javier Palma-Guerrero +14 more
TL;DR: AvrStb6 is the first avirulence gene to be functionally validated in Z. tritici, contributing to the understanding ofAvirulence in apoplastic pathogens and the mechanisms underlying GFG interactions between Z.Tritici and wheat.
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The Genome Biology of Effector Gene Evolution in Filamentous Plant Pathogens.
Andrea Sánchez-Vallet,Simone Fouché,Isabelle Fudal,Fanny E. Hartmann,Jessica L Soyer,Aurélien Tellier,Daniel Croll +6 more
TL;DR: It is concluded that effective pathogen containment strategies require a thorough understanding of the effector genome biology and the pathogen's potential for rapid adaptation.
Journal ArticleDOI
A fungal wheat pathogen evolved host specialization by extensive chromosomal rearrangements.
TL;DR: It is demonstrated that rapid turnover in the chromosomal structure of a pathogen can drive host specialization, and the main locus associated with virulence encoded a highly expressed, small secreted protein.
Journal ArticleDOI
Pangenome analyses of the wheat pathogen Zymoseptoria tritici reveal the structural basis of a highly plastic eukaryotic genome.
TL;DR: The pangenome construction of a highly polymorphic eukaryotic pathogen showed that a single reference genome significantly underestimates the gene space of a species, and the substantial accessory genome provides a cradle for adaptive evolution.
Journal ArticleDOI
A fungal avirulence factor encoded in a highly plastic genomic region triggers partial resistance to septoria tritici blotch.
Lukas Meile,Daniel Croll,Patrick C. Brunner,Clémence Plissonneau,Clémence Plissonneau,Fanny E. Hartmann,Bruce A. McDonald,Andrea Sánchez-Vallet +7 more
TL;DR: It is demonstrated that quantitative resistance and gene‐for‐gene interactions are not mutually exclusive, and localising avirulence genes in highly plastic genomic regions probably facilitates accelerated evolution that enables escape from recognition by resistance proteins.