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Fanny E. Hartmann

Researcher at Université Paris-Saclay

Publications -  38
Citations -  1561

Fanny E. Hartmann is an academic researcher from Université Paris-Saclay. The author has contributed to research in topics: Genome & Population. The author has an hindex of 15, co-authored 36 publications receiving 1000 citations. Previous affiliations of Fanny E. Hartmann include ETH Zurich & University of Paris.

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A small secreted protein in Zymoseptoria tritici is responsible for avirulence on wheat cultivars carrying the Stb6 resistance gene.

TL;DR: AvrStb6 is the first avirulence gene to be functionally validated in Z. tritici, contributing to the understanding ofAvirulence in apoplastic pathogens and the mechanisms underlying GFG interactions between Z.Tritici and wheat.
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The Genome Biology of Effector Gene Evolution in Filamentous Plant Pathogens.

TL;DR: It is concluded that effective pathogen containment strategies require a thorough understanding of the effector genome biology and the pathogen's potential for rapid adaptation.
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A fungal wheat pathogen evolved host specialization by extensive chromosomal rearrangements.

TL;DR: It is demonstrated that rapid turnover in the chromosomal structure of a pathogen can drive host specialization, and the main locus associated with virulence encoded a highly expressed, small secreted protein.
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Pangenome analyses of the wheat pathogen Zymoseptoria tritici reveal the structural basis of a highly plastic eukaryotic genome.

TL;DR: The pangenome construction of a highly polymorphic eukaryotic pathogen showed that a single reference genome significantly underestimates the gene space of a species, and the substantial accessory genome provides a cradle for adaptive evolution.
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A fungal avirulence factor encoded in a highly plastic genomic region triggers partial resistance to septoria tritici blotch.

TL;DR: It is demonstrated that quantitative resistance and gene‐for‐gene interactions are not mutually exclusive, and localising avirulence genes in highly plastic genomic regions probably facilitates accelerated evolution that enables escape from recognition by resistance proteins.