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Fathi Farag Mughrabi

Researcher at Universiti Teknologi MARA

Publications -  7
Citations -  58

Fathi Farag Mughrabi is an academic researcher from Universiti Teknologi MARA. The author has contributed to research in topics: Omeprazole & Gastric mucosa. The author has an hindex of 4, co-authored 7 publications receiving 53 citations. Previous affiliations of Fathi Farag Mughrabi include University of Benghazi & Applied Science Private University.

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Enhancement of gastric ulcer by Areca catechu nut in ethanol-induced gastric mucosal injuries in rats

TL;DR: The present finding suggests that A. catechu nut extract enhanced ulcer production as ascertained grossly by significant increasing ulcer area, and histologically by comparatively increases in ulcer areas compared to ulcer control group.
Journal Article

Effect of bis [benzyl N'-(indol-3-ylmethylene)-hydrazinecarbodithioato]-zinc(II) derivatives on wound healing in Sprague Dawley rats.

TL;DR: Results of this study showed that wounds dressed with 100 mg/ml of BHCZ significantly enhanced acceleration of the rate of wound healing enclosure, and histology of healed wounds showed comparatively less macrophages and more collagen with angiogenesis.
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Acceleration of wound healing activity by Polygonatum odoratum leaf extract in rats

TL;DR: Wounds dressed with leaf extracts significantly enhanced the acceleration of wound healing enclosure in rats and showed comparatively less scar width at wound closure and healed wound contained less inflammatory cells and more collagen with angiogenesis as compared to wounds dressed with vehicle.
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Cytoprotective Effect of Benzyl N'-(5-Chloro-indol-3-yl-methylidene)-hydrazinecarbodithioate Against Ethanol-Induced Gastric Mucosal Injury in Rats

TL;DR: The present finding suggests that benzyl N'-(5-chloroindol-3-ylmethylidene)hydrazinecarbodithioate promotes ulcer protection as ascertained by the comparative decreases in ulcer areas, reduction of edema and leucocytes infiltration of the submucosal layer.
Journal ArticleDOI

Cytoprotective effect of Benzyl N-(indol-3-ylmethylidene)-hydrazinecarbodithioate against ethanol-induced gastric mucosal injury in rats

TL;DR: The present finding suggests that Benzyl N'-(indol-3-ylmethylidene)-hydrazinecarbodithioate derivative promotes ulcer protection as ascertained by the comparative decreases in ulcer areas, reduction of edema and leucocytes infiltration of the submucosal layer.