Franklin A. Neva

TL;DR: It is suggested that IL-10, a potent, pleiotropic suppressor of all known microbicidal effector functions of macrophages, may contribute to the pathogenesis of kala-azar by inhibiting the cytokine-mediated activation of host Macrophages that is necessary for the control of leishmanial infection.

TL;DR: LIPS addresses several limitations of current ELISAs and represents a major advance in the diagnosis of S. stercoralis infection.

TL;DR: Costa Rican Lutzomyia longipalpis, vectors of non-ulcerative cutaneous disease, have very low vasodilatory activity and very little maxadilan, but their saliva strongly enhances cutaneous proliferation of Leishmania infections, suggesting that species of Lutzomers differ in their propensity to modulate the pathology of the disease they transmit.

TL;DR: It is confirmed that L tropica is a co-endemic agent of visceral leishmaniasis in India, and may shed light on the rising frequency of therapeutic unresponsiveness to sodium antimony gluconate, which complicates treatment of this lethal disease.

TL;DR: The results substantiate the role of proteases as virulence factors in strongyloidiasis, as well as other related parasitic infections, and suggest new approaches to therapy.