scispace - formally typeset
F

Frederica P. Perera

Researcher at Columbia University

Publications -  411
Citations -  33903

Frederica P. Perera is an academic researcher from Columbia University. The author has contributed to research in topics: Environmental exposure & Pregnancy. The author has an hindex of 95, co-authored 389 publications receiving 29553 citations. Previous affiliations of Frederica P. Perera include Icahn School of Medicine at Mount Sinai & Natural Resources Defense Council.

Papers
More filters
Journal ArticleDOI

Prenatal Exposure, Maternal Sensitization, and Sensitization In Utero To Indoor Allergens in an Inner-City Cohort

TL;DR: In utero sensitization to multiple indoor antigens is common, occurs to a different degree than maternal sensitization, and may involve IL-5 upregulation.
Journal ArticleDOI

Biomarkers of Environmental Tobacco Smoke in Preschool Children and Their Mothers

TL;DR: ETS exposure of young children via their mothers' smoking is associated with increases not only in the internal dose of cotinine, which has been previously reported, but also in the biologically effective dose of the carcinogenic (PAH) components of ETS ( PAH-albumin adducts).
Journal ArticleDOI

PAH-DNA adducts in cord blood and fetal and child development in a Chinese cohort.

TL;DR: In this paper, the relationship between prenatal PAH exposure and fetal and child growth and development in Tongliang, China, where a seasonally operated coal-fired power plant was the major pollution source was examined.
Journal ArticleDOI

Molecular Epidemiology: On the Path to Prevention?

TL;DR: Progress toward that goal is evaluated by using as examples well-studied environmental exposures-i.e., tobacco smoke, polycyclic aromatic hydrocarbons, aflatoxin B(1), benzene, and hepatitis B virus-and their roles in lung, breast, and liver cancers and leukemia.
Journal ArticleDOI

Prenatal acetaminophen exposure and risk of wheeze at age 5 years in an urban low-income cohort

TL;DR: Prenatal exposure to acetaminophen predicted wheeze at age 5 years in an inner-city minority cohort and the risk was modified by a functional polymorphism in GSTP1, suggesting a mechanism involving the glutathione pathway.