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Frederica P. Perera

Researcher at Columbia University

Publications -  411
Citations -  33903

Frederica P. Perera is an academic researcher from Columbia University. The author has contributed to research in topics: Environmental exposure & Pregnancy. The author has an hindex of 95, co-authored 389 publications receiving 29553 citations. Previous affiliations of Frederica P. Perera include Icahn School of Medicine at Mount Sinai & Natural Resources Defense Council.

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Molecular epidemiology and prevention of cancer.

TL;DR: Recent molecular epidemiologic studies that have focused on environmental carcinogenesis and environment-host interactions should be useful in early identification of at risk individuals and in designing and monitoring interventions (smoking cessation, exposure reduction, and chemoprevention).
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Cost of Developmental Delay from Prenatal Exposure to Airborne Polycyclic Aromatic Hydrocarbons

TL;DR: The annual costs of preschool special education services for low-income NYC children with developmental delay due to PAH exposure using the Environmentally Attributable Fraction method is estimated to be over $13.7 million per year.
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Lack of Associations among Cancer and Albumin Adducts, ras p21 Oncoprotein Levels, and CYP1A1, CYP2D6, NAT1, and NAT2 in a Nested Case-Control Study of Lung Cancer within the Physicians' Health Study

TL;DR: A “nested” case-control study within the Physicians' Health Study, a randomized trial of aspirin and BC at relatively modest doses in which baseline (enrollment/untreated) blood samples from incident cases and matched controls were selected from the bank of >14,000 stored blood samples.
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Significant interactions between maternal PAH exposure and haplotypes in candidate genes on B(a)P-DNA adducts in a NYC cohort of non-smoking African-American and Dominican mothers and newborns

TL;DR: These novel findings highlight differences in maternal and newborn genetic contributions to B[a]P-DNA adduct formation, as well as ethnic differences in gene-environment interactions, and have the potential to identify at-risk subpopulations who are susceptible to the carcinogenic potential of B[ a]P.
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Lung function growth trajectories in non-asthmatic children aged 4-9 in relation to prenatal exposure to airborne particulate matter and polycyclic aromatic hydrocarbons - Krakow birth cohort study.

TL;DR: It is indicated that in non‐asthmatic children high prenatal exposure to airborne PM2.5 and PAH is associated with lower trajectories of FVC and FEV1, but not the rate of increase over time, suggesting that the initial effect is not diminishing in time.