G
G Van den Berghe
Researcher at Katholieke Universiteit Leuven
Publications - 146
Citations - 5569
G Van den Berghe is an academic researcher from Katholieke Universiteit Leuven. The author has contributed to research in topics: Intensive care & Insulin. The author has an hindex of 34, co-authored 140 publications receiving 5080 citations. Previous affiliations of G Van den Berghe include Université catholique de Louvain & Catholic University of Leuven.
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Journal ArticleDOI
Phosphorylation and activation of heart PFK-2 by AMPK has a role in the stimulation of glycolysis during ischaemia.
Anne-Sophie Marsin,Luc Bertrand,Mark H. Rider,Johan Deprez,Christophe Beauloye,Marie-Françoise Vincent,G Van den Berghe,David Carling,Louis Hue +8 more
TL;DR: AMPK phosphorylates and activates heart PFK-2 in vitro and in intact cells, and is likely to be involved in the stimulation of heart glycolysis during ischaemia.
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Insulin therapy protects the central and peripheral nervous system of intensive care patients
TL;DR: Preventing even moderate hyperglycemia with insulin during intensive care protected the central and peripheral nervous systems, with clinical consequences such as shortening of intensive care dependency and possibly better long-term rehabilitation.
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Introductory to the ESPEN Guidelines on Enteral Nutrition: Terminology, Definitions and General Topics
Herbert Lochs,Simon Allison,Rémy Meier,Matthias Pirlich,Jens Kondrup,S. Schneider,G Van den Berghe,Claude Pichard +7 more
TL;DR: In this chapter terms and definitions used throughout all guidelines are explained and more general questions, which might be important in most indications are dealt with, i.e. use of fibre containing and diabetes formulae.
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The Sick and the Weak: Neuropathies/Myopathies in the Critically Ill
Oliver Friedrich,Michael B. Reid,G Van den Berghe,Ilse Vanhorebeek,Greet Hermans,Mark M. Rich,Lars Larsson +6 more
TL;DR: A classification of different ICUAW forms (CIM, CIP, sepsis-induced, steroid-denervation myopathy) and pathophysiological mechanisms from clinical and animal model data are proposed and highlighted, allowing separation of risk factors that may trigger distinct mechanisms contributing to weakness.
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Novel insights into the neuroendocrinology of critical illness
TL;DR: A uniformly reduced pulsatile secretion of ACTH, TSH, LH, prolactin (PRL) and GH has been observed in protracted critical illness, causing diminished stimulation of several target organs and metabolic improvement was noted with the combined infusion of GHRP and TRH.