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Gayle M. Gordillo
Researcher at Indiana University
Publications - 88
Citations - 6305
Gayle M. Gordillo is an academic researcher from Indiana University. The author has contributed to research in topics: Wound healing & Endothelial stem cell. The author has an hindex of 29, co-authored 78 publications receiving 5338 citations. Previous affiliations of Gayle M. Gordillo include The Ohio State University Wexner Medical Center & Ohio State University.
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Oral Administration of Blueberry Inhibits Angiogenic Tumor Growth and Enhances Survival of Mice with Endothelial Cell Neoplasm
TL;DR: This work provides first evidence demonstrating that BBE can limit tumor formation through antiangiogenic effects and inhibition of JNK and NF-kappaB signaling pathways.
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Preclinical Models of Wound Healing: Is Man the Model? Proceedings of the Wound Healing Society Symposium
Gayle M. Gordillo,Stéphanie F. Bernatchez,Robert F. Diegelmann,Luisa A. Di Pietro,Elof Eriksson,Boris Hinz,Harriet W. Hopf,Robert S. Kirsner,Paul Y. Liu,Laura K.S. Parnell,George E. Sandusky,Chandan K. Sen,Marjana Tomic-Canic,Susan W. Volk +13 more
TL;DR: This consensus statement issued by the Wound Healing Society symposium strongly encourages all wound researchers to involve human wound tissue validation studies to make their animal and cell biology studies more translationally and clinically significant.
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Nox-4-dependent nuclear H2O2 drives DNA oxidation resulting in 8-OHdG as urinary biomarker and hemangioendothelioma formation.
TL;DR: It is reported that in EOMA cells, nox-4 is localized such that it delivers H2O2 to the nuclear compartment, which causes oxidative modification of DNA, which can be detected in the urine of tumor-bearing mice as 8-hydroxy-2-deoxyguanosine.
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Wound healing society 2015 update on guidelines for pressure ulcers: Guidelines for the treatment of pressure ulcers
Lisa J. Gould,Mike Stuntz,Michelle Giovannelli,Amier Ahmad,Rummana Aslam,Margaret Mullen-Fortino,JoAnne D. Whitney,Jason Calhoun,Robert S. Kirsner,Gayle M. Gordillo +9 more
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A key angiogenic role of monocyte chemoattractant protein-1 in hemangioendothelioma proliferation.
Gayle M. Gordillo,Duygu Onat,Michael P. Stockinger,Sashwati Roy,Mustafa Atalay,F. Michael Beck,Chandan K. Sen +6 more
TL;DR: In vivo evidence of a complete response for any neoplasm, specifically a vascular proliferative lesion, to anti-MCP-1 therapy in animals with intact immune systems is produced, presenting the first evidence that M CP-1 is required for HE proliferation and may promote the growth of these lesions by stimulating angiogenic behavior of endothelial cells.