George Dimitriadis

TL;DR: These insulin effects serve to encourage the synthesis of carbohydrate, fat and protein, therefore, insulin can be considered to be an anabolic hormone.

TL;DR: In patients with HO and SHO, insulin resistance was comparable and insulin-stimulated rates of glucose transport in isolated monocytes were decreased due to impaired translocation of GLUT4 glucose transporters on the plasma membrane, which could justify the increased risk for insulin resistance-associated disorders, such as cardiovascular disease, observed in patients withHO or SHO.

TL;DR: Amylin may be a factor in the etiology of the insulin resistance in type 2 diabetes mellitus, as both deposition of the peptide in islet amyloid and decreased rates of glucose uptake and glycogen synthesis in skeletal muscle are characteristic of this condition.

TL;DR: A redistribution of glucose away from the pathway of glycogen synthesis and glucose oxidation could maintain a normal rate of lactate formation although the rate of glucose transport is decreased, and the changes in the sensitivity of glucose phosphorylation to insulin seen under these conditions are secondary to those in glucose transport.

TL;DR: The objective was to examine the effects of activation by Phorbol 12‐myristate 13‐acetate (PMA) or lipopolysaccharide (LPS) and insulin on the expression of GLUT isoforms in all subpopulations of WBC.