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George J. F. Heigenhauser

Researcher at McMaster University

Publications -  278
Citations -  18003

George J. F. Heigenhauser is an academic researcher from McMaster University. The author has contributed to research in topics: Skeletal muscle & Glycogen. The author has an hindex of 77, co-authored 278 publications receiving 17027 citations. Previous affiliations of George J. F. Heigenhauser include University of Ljubljana & McMaster University Medical Centre.

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Six sessions of sprint interval training increases muscle oxidative potential and cycle endurance capacity in humans

TL;DR: It is concluded that short sprint interval training (approximately 15 min of intense exercise over 2 wk) increased muscle oxidative potential and doubled endurance capacity during intense aerobic cycling in recreationally active individuals.
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Repeated transient mRNA bursts precede increases in transcriptional and mitochondrial proteins during training in human skeletal muscle

TL;DR: The training‐induced increases in transcriptional and mitochondrial proteins appear to result from the cumulative effects of transient bursts in their mRNAs, and training-induced mitochondrial biogenesis appears to involve re‐modelling in addition to increased mitochondrial content.
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Two weeks of high-intensity aerobic interval training increases the capacity for fat oxidation during exercise in women

TL;DR: It is shown that seven sessions of HIIT over 2 wk induced marked increases in whole body and skeletal muscle capacity for fatty acid oxidation during exercise in moderately active women.
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Effect of short-term sprint interval training on human skeletal muscle carbohydrate metabolism during exercise and time-trial performance

TL;DR: It is concluded that short-term SIT improved cycling TT performance and resulted in a closer matching of glycogenolytic flux and pyruvate oxidation during submaximal exercise.
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Triacylglycerol accumulation in human obesity and type 2 diabetes is associated with increased rates of skeletal muscle fatty acid transport and increased sarcolemmal FAT/CD36.

TL;DR: FAT/CD36 provides another potential therapeutic target for the prevention and/or treatment of insulin resistance and the increased rate of fatty acid transport may contribute to the increased rates of triacylglycerol accumulation in human skeletal muscle.