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Gerald Siegwart

Researcher at University of Natural Resources and Life Sciences, Vienna

Publications -  5
Citations -  321

Gerald Siegwart is an academic researcher from University of Natural Resources and Life Sciences, Vienna. The author has contributed to research in topics: Gene & Quantitative trait locus. The author has an hindex of 5, co-authored 5 publications receiving 265 citations.

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Transcriptomic characterization of two major Fusarium resistance quantitative trait loci (QTLs), Fhb1 and Qfhs.ifa‐5A, identifies novel candidate genes

TL;DR: A uridine diphosphate (UDP)‐glycosyltransferase gene, designated TaUGT12887, exhibiting a positive difference in response to the pathogen in lines harbouring both QTLs relative to lines carrying only the Qfhs.ifa‐5A resistance allele is identified, suggesting Fhb1 dependence of this transcript.
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Quantitative trait loci-dependent analysis of a gene co-expression network associated with Fusarium head blight resistance in bread wheat ( Triticum aestivum L.)

TL;DR: A combination of a network-driven approach and differential gene expression analysis identified genes and pathways associated with Fhb1 and Qfhs, which take prominent roles in the pathogen response.
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Joint Transcriptomic and Metabolomic Analyses Reveal Changes in the Primary Metabolism and Imbalances in the Subgenome Orchestration in the Bread Wheat Molecular Response to Fusarium graminearum

TL;DR: Transcriptomics and metabolomics data are integrated to dissect the molecular response to the fungus and its main virulence factor, the toxin deoxynivalenol in near-isogenic lines segregating for two resistance quantitative trait loci, Fhb1 and Qfhs, and identify several genes that may contribute to increased pathogen endurance.
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Hydrophilic interaction liquid chromatography coupled with tandem mass spectrometry for the quantification of uridine diphosphate-glucose, uridine diphosphate-glucuronic acid, deoxynivalenol and its glucoside: In-house validation and application to wheat.

TL;DR: It is proposed that the higher ability to detoxify DON into DON-3-Glc might be a consequence of the higher cellular UDP-glucose pool in the resistant cultivar, showing different resistance to the severe plant disease Fusarium head blight.