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Gerhard Fusch

Researcher at McMaster University

Publications -  83
Citations -  2007

Gerhard Fusch is an academic researcher from McMaster University. The author has contributed to research in topics: Breast milk & Medicine. The author has an hindex of 24, co-authored 74 publications receiving 1665 citations. Previous affiliations of Gerhard Fusch include McMaster University Medical Centre & University of Greifswald.

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Target fortification of breast milk with fat, protein, and carbohydrates for preterm infants.

TL;DR: Daily target fortification in breast milk can be safely implemented in clinical routine and may improve growth and the linear relationship between milk intake and weight gain observed in study babies but not seen in matched controls may be related to the variable composition of breast milk.
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Rapid measurement of macronutrients in breast milk: How reliable are infrared milk analyzers?

TL;DR: Two milk analyzers for breast milk analysis with reference methods are validated and the potential to be introduced in clinical routine to measure fat and protein content, but will need major adjustments.
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Psychological Stress-Induced, IDO1-Dependent Tryptophan Catabolism: Implications on Immunosuppression in Mice and Humans

TL;DR: Psychological stress stimulates cytokine-driven IDO1 activation and Trp depletion which seems to have a central role for developing stress-induced immunosuppression and behavioral alteration.
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Prevalence and frequency of circulating t(14;18)-MBR translocation carrying cells in healthy individuals.

TL;DR: The age dependent prevalence and frequency of BCL2/IgH rearrangements in 715 healthy individuals ranging from newborns to octo‐ and nonagenarians will further improve the understanding of the relevance of t(14;18)‐positive cells in healthy individuals as a risk marker toward the development into lymphoma precursors.
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Hypermetabolic syndrome as a consequence of repeated psychological stress in mice.

TL;DR: Repeated stressed mice developed a hypermetabolic syndrome with the severe loss of lean body mass, hyperglycemia, dyslipidemia, increased amino acid turnover, and acidosis, leading to a drastic reduction of the individual's energy reserves.