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Giorgio Battaglia

Researcher at University of Strathclyde

Publications -  75
Citations -  3677

Giorgio Battaglia is an academic researcher from University of Strathclyde. The author has contributed to research in topics: Cortical dysplasia & Epilepsy. The author has an hindex of 26, co-authored 74 publications receiving 3258 citations.

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Selective vulnerability of spinal and cortical motor neuron subpopulations in delta7 SMA mice.

TL;DR: Investigating the spinal cord and cerebral motor cortex of ∆7 SMA mice during development found progressive post-natal loss of spinal motor neurons, and a higher vulnerability of motor neurons innervating proximal and axial muscles, suggesting that SMA pathogenesis is likely more complex than previously anticipated.
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Status epilepticus-induced pathologic plasticity in a rat model of focal cortical dysplasia

TL;DR: Data show that status epilepticus sets in motion a pathological process capable of significantly changing the cellular and molecular features of pre-existing experimental cortical malformations, and suggest that seizure recurrence in human focal cortical dysplasia might be an additional factor in establishing a pathological circuitry that favours chronic neuronal hyperexcitability.
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Flexible endoscopic treatment for Zenker's diverticulum with the SB Knife. Preliminary results from a single-center experience

TL;DR: The experience with a recently designed, monopolar, rotating, scissor‐shaped device (SB Knife) for Zenker's diverticulum (ZD) is reported.
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Management of esophageal cancer in patients aged over 80 years.

TL;DR: Even in octogenarian patients--with limited comorbidities and fit for surgery--esophagectomy may be regarded as a valid treatment option, although this remains possible only in a small minority of 80-90-year old patients.
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Epileptic and imaging findings in perinatal hypoxic-ischemic encephalopathy with ulegyria

TL;DR: The electroclinical features of hypoxic-ischemic encephalopathy when ulegyria is one of its main neuropathological features are characterized, and epilepsy was a prominent component and drug refractoriness was an almost constant correlate of this form of symptomatic epilepsy.