G
Giorgio Battaglia
Researcher at University of Strathclyde
Publications - 75
Citations - 3677
Giorgio Battaglia is an academic researcher from University of Strathclyde. The author has contributed to research in topics: Cortical dysplasia & Epilepsy. The author has an hindex of 26, co-authored 74 publications receiving 3258 citations.
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Journal ArticleDOI
A germline predictive signature of response to platinum chemotherapy in esophageal cancer.
Enrica Rumiato,Elisa Boldrin,Sandro Malacrida,Giorgio Battaglia,Paolo Bocus,Carlo Castoro,Matteo Cagol,Vanna Chiarion-Sileni,Alberto Ruol,Alberto Amadori,Daniela Saggioro +10 more
TL;DR: A genetic signature of response to platinum-based neoadjuvant chemotherapy in EC patients is identified and the potential benefit of combining genetic and clinical variables for personalized EC management is disclosed.
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Continuous neurodegeneration and death pathway activation in neurons and glia in an experimental model of severe chronic epilepsy.
Paola Nobili,Francesca Colciaghi,Adele Finardi,Sara Zambon,Denise Locatelli,Giorgio Battaglia +5 more
TL;DR: Data support the content that at least in the MP rat model the recurrence of seizures can also sustain cell death mechanisms, thus continuously contributing to the pathologic process triggered by the occurrence of SE.
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Selective vulnerability of spinal and cortical motor neuron subpopulations in delta7 sma mice.
Marina Boido,Paolo d'Errico,Antonio Piras,Valeria Valsecchi,E. De Amicis,Denise Locatelli,Silvia Capra,F. Vagni,Giorgio Battaglia,Alessandro Vercelli +9 more
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Detection of genetic alterations in cfDNA as a possible strategy to monitor the neoplastic progression of Barrett's esophagus.
Enrica Rumiato,Elisa Boldrin,Sandro Malacrida,Stefano Realdon,Matteo Fassan,T. Morbin,Giorgio Battaglia,Alberto Amadori,Massimo Rugge,Daniela Saggioro +9 more
TL;DR: In the cfDNA of dysplastic Be patients, the frequency of genetic alterations is statistically higher than that of metaplastic BE patients, suggesting that the use of liquid biopsy could become a minimally invasive diagnostic tool to implement BE patient monitoring.
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Spinal muscular atrophy pathogenic mutations impair the axonogenic properties of axonal-survival of motor neuron.
Denise Locatelli,Paolo d'Errico,Silvia Capra,Adele Finardi,Francesca Colciaghi,Veronica Setola,Mineko Terao,Enrico Garattini,Giorgio Battaglia +8 more
TL;DR: In this paper, the authors investigated whether SMA pathogenic mutations located in the N-terminal part of the SMN1 protein affected a-SMN function and found that Tudor domain mutations are functionally relevant not only for FL SMN but also for a- SMN, raising the possibility that also SMN loss of function may contribute to the pathogenic steps leading to SMA.