G
Glenn K. McConell
Researcher at Victoria University, Australia
Publications - 151
Citations - 6509
Glenn K. McConell is an academic researcher from Victoria University, Australia. The author has contributed to research in topics: Skeletal muscle & Glucose uptake. The author has an hindex of 46, co-authored 149 publications receiving 6026 citations. Previous affiliations of Glenn K. McConell include Monash University, Clayton campus & Royal Melbourne Hospital.
Papers
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Journal ArticleDOI
AMPK signaling in contracting human skeletal muscle: acetyl-CoA carboxylase and NO synthase phosphorylation.
Zhi-Ping Chen,Glenn K. McConell,Belinda J. Michell,Rodney J. Snow,Benedict J. Canny,Bruce E. Kemp +5 more
TL;DR: Observations support the concept that inhibition of ACC is an important component in stimulating fatty acid oxidation in response to exercise and that there is coordinated regulation of nNOSmu to protect the muscle from ischemia/metabolic stress.
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Effect of Exercise Intensity on Skeletal Muscle AMPK Signaling in Humans
Zhi-Ping Chen,Terry J. Stephens,Sid Murthy,Benedict J. Canny,Mark Hargreaves,Lee A. Witters,Bruce E. Kemp,Glenn K. McConell +7 more
TL;DR: Results indicate that ACC beta phosphorylation is especially sensitive to exercise and tightly coupled to AMPK signaling and that AMPK activation does not depend on AMPK kinase activation during exercise.
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Nitric oxide synthase inhibition reduces leg glucose uptake but not blood flow during dynamic exercise in humans.
TL;DR: It is indicated that NO production contributes substantially to exercise-mediated skeletal muscle glucose uptake in humans independent of skeletal muscle blood flow.
Journal ArticleDOI
Influence of muscle glycogen on glycogenolysis and glucose uptake during exercise in humans
TL;DR: It is indicated that preexercise muscle glycogen availability influences muscle glycogeneolysis, but not glucose uptake, during exercise, as shown in the results.
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Type 2 diabetic individuals have impaired leg blood flow responses to exercise: role of endothelium-dependent vasodilation.
TL;DR: The increase in LBF during exercise is substantially attenuated in type 2 diabetic compared with matched control subjects, and this mechanism may be of importance in determining the leg ischemic threshold in diabetic individuals with peripheral vascular disease.