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Gloria DeGrandi-Hoffman

Researcher at Agricultural Research Service

Publications -  115
Citations -  3764

Gloria DeGrandi-Hoffman is an academic researcher from Agricultural Research Service. The author has contributed to research in topics: Honey bee & Population. The author has an hindex of 31, co-authored 110 publications receiving 3050 citations. Previous affiliations of Gloria DeGrandi-Hoffman include United States Department of Agriculture & University of Arizona.

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Honey bee aggression supports a link between gene regulation and behavioral evolution

TL;DR: It is shown that aggression-related genes with inherited patterns of brain expression are also environmentally regulated, and it appears that one element in the evolution of different degrees of aggressive behavior in honey bees involved changes in regulation of genes that mediate the response to alarm pheromone.
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The effect of diet on protein concentration, hypopharyngeal gland development and virus load in worker honey bees (Apis mellifera L.).

TL;DR: Overall results suggest a connection between diet, protein levels and immune response and indicate that colony losses might be reduced by alleviating protein stress through supplemental feeding.
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THE AFRICAN HONEY BEE: Factors Contributing to a Successful Biological Invasion*

TL;DR: This work provides a synthesis of recent research on the African bee, concentrating on its ability to displace European honey bees, and considers the genetic composition of the expanding population and the symmetry of gene flow between African and European bees.
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An emerging paradigm of colony health: microbial balance of the honey bee and hive ( Apis mellifera )

TL;DR: It is argued that the honey bee should be viewed as a model system to examine the effect of microbial communities on host nutrition and pathogen defense, and the niche requirements and maintenance of beneficial honey bee symbionts are largely unknown.
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Nutrition, immunity and viral infections in honey bees.

TL;DR: This work discusses how honey bees decrease the risk of disease outbreaks by a combination of behaviors (social immunity) and individual immune function and a relationship between the effectiveness of social and individual immunity and the nutritional state of the colony.