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Guido Nicolò

Researcher at National Cancer Research Institute

Publications -  57
Citations -  2900

Guido Nicolò is an academic researcher from National Cancer Research Institute. The author has contributed to research in topics: Sentinel lymph node & Breast cancer. The author has an hindex of 29, co-authored 57 publications receiving 2813 citations. Previous affiliations of Guido Nicolò include European Institute of Oncology.

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The fibronectin isoform containing the ED-B oncofetal domain: a marker of angiogenesis.

TL;DR: It is demonstrated here, using immunohistochemical techniques, that while this FN isoform is undetectable in mature vessels, it is highly expressed during angiogenesis both in neoplastic and in normal tissues, as in the case of the functional layer of endometrium during the proliferative phase.
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Structure of the Supernumerary Ring and Giant Rod Chromosomes in Adipose Tissue Tumors

TL;DR: This work extensively investigated the structure and composition of rings and giant rods in a series of 17 WDLPS‐ALP samples and three intra‐ or intermuscular lipomas (IMLP), revealing a unique combination of particular features strikingly related to these tumors.
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Endoscopic retrograde forceps biopsy and brush cytology of biliary strictures: a prospective study

TL;DR: Endoscopic retrograde brush cytology alone may be sufficient in daily practice, at least in centers that have access to experienced cytopathologists, and use of forceps biopsy in selected cases where Brush cytology is negative.
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Expression of different tenascin isoforms in normal, hyperplastic and neoplastic human breast tissues.

TL;DR: The data reported here suggest that, in the breast, expression of the high‐molecular‐mass TN isoform is a marker of stromal element proliferation and that thisTN isoform could play a role in generating a permissive environment for proliferation, invasion and metastasis of neoplastic epithelial cells.
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Complex composition and co-amplification of SAS and MDM2 in ring and giant rod marker chromosomes in well-differentiated liposarcoma.

TL;DR: To begin to fathom the function of the extra abnormal chromosomes, the amplification of genes, including SAS, MDM2, and GADDI53/CHOP, known to be in the region 12q 13–14 were examined and SAS andMDM2 demonstrated constant co‐amplification.