G
Gustavo Rinaldi
Researcher at National University of La Plata
Publications - 41
Citations - 595
Gustavo Rinaldi is an academic researcher from National University of La Plata. The author has contributed to research in topics: Calcium & Nifedipine. The author has an hindex of 13, co-authored 41 publications receiving 518 citations. Previous affiliations of Gustavo Rinaldi include National Scientific and Technical Research Council & Facultad de Ciencias Médicas.
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Journal ArticleDOI
Amaranth seed protein hydrolysates have in vivo and in vitro antihypertensive activity
TL;DR: Experiments suggest that the hypotensive effect could be attributed to a lowering of the peripheral resistance, and it is assumed that the amaranth hydrolysates would be acting at the level of the local or autocrine renin–angiotensin system (RAS).
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Increased intracellular Ca2+ and SR Ca2+ load contribute to arrhythmias after acidosis in rat heart. Role of Ca2+/calmodulin-dependent protein kinase II.
María Matilde Said,Romina Valeria Becerra,Julieta Palomeque,Gustavo Rinaldi,Marcia A. Kaetzel,Paula L. Diaz-Sylvester,J. A. Copello,John R. Dedman,Cecilia Mundiña-Weilenmann,Leticia Vittone,Alicia Mattiazzi +10 more
TL;DR: The results reveal that arrhythmias after acidosis are dependent on CaMKII activation and are associated with an increase in SR Ca(2+) load, which appears to be mainly due to the increase in PT-PLN.
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CaMKII-dependent phosphorylation of cardiac ryanodine receptors regulates cell death in cardiac ischemia/reperfusion injury.
Mariano Nahuel Di Carlo,Matilde Said,Haiyun Ling,Carlos Alfredo Valverde,Verónica Celeste De Giusti,Leandro Matías Sommese,Julieta Palomeque,Ernesto A. Aiello,Darlene G. Skapura,Gustavo Rinaldi,Jonathan L. Respress,Joan Heller Brown,Xander H.T. Wehrens,Margarita Ana Salas,Alicia Mattiazzi +14 more
TL;DR: A major role is revealed for the phosphorylation of S2814 site on RyR2 in CaMKII-dependent I/R cardiac damage and it is shown that CaMK II-dependent increase in PLN phosphorylated during reperfusion opposes rather than contributes to I/ R damage.
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Antioxidant capacity of Maillard reaction products in the digestive tract: An in vitro and in vivo study
TL;DR: The in vitro digestion/fermentation procedure combined with ICRED determination would be a useful methodology to predict the release of antioxidant MRP in vivo.
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Interaction between calcium and hydrogen ionsin canine coronary arteries
TL;DR: The data do not support the hypothesis of hydrogen ion changes inducing calcium influx alterations in a competitive fashion as the mechanism involved to contract or relax conduit coronary arteries but suggest that acid-base variations alter the contractile activity of vascular smooth muscle by modulating Ca2+ efflux.