H
Hadi Al-Hasani
Researcher at University of Düsseldorf
Publications - 150
Citations - 4507
Hadi Al-Hasani is an academic researcher from University of Düsseldorf. The author has contributed to research in topics: Insulin resistance & GLUT4. The author has an hindex of 35, co-authored 122 publications receiving 3559 citations. Previous affiliations of Hadi Al-Hasani include National Institutes of Health & University of Cologne.
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Journal ArticleDOI
Risk of diabetes-associated diseases in subgroups of patients with recent-onset diabetes: a 5-year follow-up study.
Oana P. Zaharia,Klaus Strassburger,Alexander Strom,Gidon J. Bönhof,Yanislava Karusheva,Sofia Antoniou,Kálmán Bódis,D Markgraf,Volker Burkart,Karsten Müssig,Jong-Hee Hwang,Olof Asplund,Leif Groop,Emma Ahlqvist,Jochen Seissler,Peter P. Nawroth,Stefan Kopf,Sebastian M. Schmid,Michael Stumvoll,Andreas Pfeiffer,Stefan Kabisch,Sergey Tselmin,Hans U. Häring,Dan Ziegler,Oliver Kuss,Julia Szendroedi,Michael Roden,Bengt-Frederik Belgardt,Anette E. Buyken,Jürgen Eckel,Gerd Geerling,Hadi Al-Hasani,Christian Herder,Andrea Icks,Jorg Kotzka,Eckart Lammert,Daniel F. Markgraf,Wolfgang Rathmann +37 more
TL;DR: In patients with newly diagnosed type 1 or type 2 diabetes, hepatocellular lipid content was highest at baseline in patients assigned to the SIRD cluster and insulin resistance index at baseline was highest in patients with SIRD and MOD.
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Tbc1d1 mutation in lean mouse strain confers leanness and protects from diet-induced obesity.
Alexandra Chadt,Katja Leicht,Atul S. Deshmukh,Lake Q. Jiang,Stephan Scherneck,Ulrike Bernhardt,T. Dreja,Heike Vogel,K. Schmolz,Reinhart Kluge,Juleen R. Zierath,Claus Hultschig,Rob C. Hoeben,Annette Schürmann,Hans-Georg Joost,Hadi Al-Hasani +15 more
TL;DR: Mutation of TBC1d1 suppresses high-fat diet–induced obesity by increasing lipid use in skeletal muscle by results in a truncated protein lacking the TBC Rab–GTPase-activating protein domain.
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Dehydroascorbic Acid Transport by GLUT4 in XenopusOocytes and Isolated Rat Adipocytes
Steven C. Rumsey,Rushad Daruwala,Hadi Al-Hasani,Mary Jane Zarnowski,Ian A. Simpson,Mark Levine +5 more
TL;DR: It is indicated that the insulin-sensitive transporter GLUT4 transports DHA in both rat adipocytes and Xenopus oocytes, and Alterations of this mechanism in diabetes could have clinical implications for ascorbate utilization.
Journal ArticleDOI
Glucose transporters in adipose tissue, liver, and skeletal muscle in metabolic health and disease
Alexandra Chadt,Hadi Al-Hasani +1 more
TL;DR: An array of circulating metabolites and hormone-like molecules and potential supplementary glucose transporters play roles in fine-tuning glucose flux between the different organs in response to an altered energy demand.
Journal ArticleDOI
Opposite translational control of GLUT1 and GLUT4 glucose transporter mRNAs in response to insulin. Role of mammalian target of rapamycin, protein kinase b, and phosphatidylinositol 3-kinase in GLUT1 mRNA translation.
TL;DR: The opposite effects of insulin on GLUT1 and GLUT4 mRNA translation are unraveled, and increased GLut1 mRNA translation appears to occur via the PI3K/PKB/mTOR/4E-BP1 cascade.