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Haibo Zhao

Researcher at Huazhong University of Science and Technology

Publications -  289
Citations -  11632

Haibo Zhao is an academic researcher from Huazhong University of Science and Technology. The author has contributed to research in topics: Chemical looping combustion & Combustion. The author has an hindex of 51, co-authored 272 publications receiving 9496 citations. Previous affiliations of Haibo Zhao include University of Turku & University of Duisburg-Essen.

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The cell biology of osteoclast function

TL;DR: Osteoclasts have developed an efficient machinery for dissolving crystalline hydroxyapatite and degrading organic bone matrix rich in collagen fibers that allows osteoclasts to remove large amounts of matrix-degradation products without losing their tight attachment to underlying bone.
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Notch signaling maintains bone marrow mesenchymal progenitors by suppressing osteoblast differentiation.

TL;DR: A model wherein Notch signaling in bone marrow normally acts to maintain a pool of mesenchymal progenitors by suppressing osteoblast differentiation is supported, whereas bone formation in vivo may be enhanced by transiently suppressing this pathway.
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Glucocorticoids suppress bone formation via the osteoclast

TL;DR: Compared with osteoclasts modulating the osteoblast-suppressive effect of DEX, GRoc-/- mice are protected from the steroid's inhibition of bone formation, suggesting that an intermediary cell transmits a component of the bone-suppression effects of GCs to osteoblasts in the intact animal.
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Carbon nanotube yarn strain sensors

TL;DR: A prototype carbon nanotube (CNT) yarn strain sensor with excellent repeatability and stability for in situ structural health monitoring was developed and showed consistent piezoresistive behavior under repetitive straining and unloading.
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The Estrogen Receptor-α in Osteoclasts Mediates the Protective Effects of Estrogens on Cancellous But Not Cortical Bone

TL;DR: It is concluded that estrogens attenuate osteoclast generation and life span via cell autonomous effects mediated by DNA-binding-independent actions of ERalpha, and elimination of these effects is sufficient for loss of bone in the cancellous compartment.