H
Hans-Peter Dienes
Researcher at Medical University of Vienna
Publications - 230
Citations - 8831
Hans-Peter Dienes is an academic researcher from Medical University of Vienna. The author has contributed to research in topics: Hepatitis & Autoimmune hepatitis. The author has an hindex of 48, co-authored 226 publications receiving 8183 citations. Previous affiliations of Hans-Peter Dienes include University of Cologne & University of Mainz.
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Journal ArticleDOI
Simplified criteria for the diagnosis of autoimmune hepatitis
E. M. Hennes,Mikio Zeniya,Albert J. Czaja,Albert Parés,George N. Dalekos,Edward L. Krawitt,Paulo Lisboa Bittencourt,Gilda Porta,Kirsten Muri Boberg,Harald Hofer,Francesco B. Bianchi,Minoru Shibata,Christoph Schramm,Barbara Eisenmann de Torres,Peter R. Galle,Ian G. McFarlane,Hans-Peter Dienes,AW Lohse +17 more
TL;DR: A reliable diagnosis of AIH can be made using a very simple diagnostic score proposed, which is the diagnosis of probable AIH at a cutoff point greater than 6 points and definite AIH 7 points or higher.
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MicroRNA gene expression profile of hepatitis C virus–associated hepatocellular carcinoma
Heike Varnholt,Uta Drebber,F Schulze,Inga Wedemeyer,Peter Schirmacher,Hans-Peter Dienes,Margarete Odenthal +6 more
TL;DR: A subset of microRNAs are aberrantly expressed in primary liver tumors, serving both as putative tumor suppressors and as oncogenic regulators.
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Expression of the gene of the α-smooth muscle-actin isoform in rat liver and in rat fat-storing (ITO) cells
G. Ramadori,Th. Veit,S. Schwögler,Hans-Peter Dienes,T. Knittel,H. Rieder,K H Meyer zum Büschenfelde +6 more
TL;DR: It is suggested thatFat storing cells in the liver are smooth muscle-related myo-fibroblasts with special functions, as α-SM-actin isoform gene expression was strongly increased in the rat liver after acute or chronic damage induced by treatment with carbon tetrachloride.
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Characterization of the overlap syndrome of primary biliary cirrhosis (PBC) and autoimmune hepatitis: Evidence for it being a hepatitic form of PBC in genetically susceptible individuals
Ansgar W. Lohse,Karl-Hermann Meyer zum Büschenfelde,Beate Franz,Stephan Kanzler,G. Gerken,Hans-Peter Dienes +5 more
TL;DR: It is suggested that therapy of PBC should be guided by the degree of biochemical and histological hepatic involvement, and the name “overlap syndrome” should be abandoned for “PBC, hepatitic form.”
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Atorvastatin attenuates hepatic fibrosis in rats after bile duct ligation via decreased turnover of hepatic stellate cells
Jonel Trebicka,Martin Hennenberg,Margarete Odenthal,Khanwali Shir,Sabine Klein,Michaela Granzow,Annabelle Vogt,Hans-Peter Dienes,Frank Lammert,Frank Lammert,Jürg Reichen,Jörg Heller,Tilman Sauerbruch +12 more
TL;DR: Prophylactic and early therapy with atorvastatin significantly attenuated fibrosis and HSC activation and led to a more quiescent state of HSC with less proliferation and apoptosis, while hepatic inflammation did not change.