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He Chang

Researcher at Niigata University

Publications -  12
Citations -  372

He Chang is an academic researcher from Niigata University. The author has contributed to research in topics: Gene delivery & T cell. The author has an hindex of 9, co-authored 12 publications receiving 356 citations. Previous affiliations of He Chang include RMIT University.

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Hydrodynamic-based delivery of an interleukin-22-Ig fusion gene ameliorates experimental autoimmune myocarditis in rats.

TL;DR: Results showed that the expression of immunologic molecules in IL-1-stimulated NC cells was significantly decreased by rIL-22 or serum containing IL-22-Ig, and the reason for this effectiveness may be that IL- 22 suppressed gene expression of PG synthases, IL-6, and chemokines in activated NC noninflammatory cells.
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Alteration of IL-17 related protein expressions in experimental autoimmune myocarditis and inhibition of IL-17 by IL-10-Ig fusion gene transfer.

TL;DR: IL-17 is highly produced by αβT cells in the early phase of EAM hearts and IL-17 inhibition might be a possible mechanism of the amelioration of E AM by IL-10-Ig treatment, suggesting that IL- 17 produced by Th17 plays an important role in the pathogenesis of rat EAM.
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Effect of Hydrodynamics-Based Gene Delivery of Plasmid DNA Encoding Interleukin-1 Receptor Antagonist-Ig for Treatment of Rat Autoimmune Myocarditis Possible Mechanism for Lymphocytes and Noncardiac Cells

TL;DR: EAM was suppressed by hydrodynamics-based delivery of plasmid DNA encoding IL-1RA-Ig and the expression of immune-relevant genes within noncardiomyocytic cells cultured from EAM hearts or concanavalin A-stimulated lymphocytes derived from lymph nodes in EAM-affected rats.
Journal ArticleDOI

Alteration of IL-17 Related Protein Expressions in Experimental Autoimmune Myocarditis and Inhibition of IL-17 by IL-10-Ig Fusion Gene Transfer

TL;DR: IL-17 produced by Th17 plays an important role in the pathogenesis of rat EAM, and IL-17 inhibition might be a possible mechanism of the amelioration of EAM by IL-10-Ig treatment.