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Henry E. Young

Researcher at Mercer University

Publications -  37
Citations -  2396

Henry E. Young is an academic researcher from Mercer University. The author has contributed to research in topics: Stem cell & Adult stem cell. The author has an hindex of 19, co-authored 37 publications receiving 2358 citations.

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Human reserve pluripotent mesenchymal stem cells are present in the connective tissues of skeletal muscle and dermis derived from fetal, adult, and geriatric donors.

TL;DR: This study details the profile of 13 cell surface cluster differentiation markers on human reserve stem cells derived from connective tissues as well as lineage‐uncommitted pluripotent stem cells capable of forming muscle, adipocytes, cartilage, bone, fibroblasts, and endothelial cells.
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Mesenchymal stem cells reside within the connective tissues of many organs

TL;DR: The results suggest that progenitor mesenchymal stem cells and putative pluripotent mesenchyl stem cells with the potential to form at least four tissues of mesodermal origin have a widespread distribution throughout the body, being located within the connective tissue compartments of many organs and organ systems.
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Adult stem cells.

TL;DR: Until recently it was generally assumed that the precursor cells in postnatal individuals were limited to lineage-committed progenitor cells specific for various tissues, but studies by Young, his colleagues, and others have demonstrated the presence of two categories of precursor cells that reside within the organs and tissues of postnatal animals.
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Human pluripotent and progenitor cells display cell surface cluster differentiation markers CD10, CD13, CD56, and MHC class-I.

TL;DR: This study examined human pluripotent and progenitor cells isolated from fetal, mature, and geriatric individuals for the possible presence of 15 CD markers, and reports the first identification of CD10, CD13, CD56, and MHC Class-I cell surface antigens on these human cells.
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Formation of Abdominal Adhesions Is Inhibited by Antibodies to Transforming Growth Factor-β1

TL;DR: Reducing levels of TGF-beta1 alone can be effective in preventing abdominal adhesions in rats, and specifically reducing levels of anti-TGF- beta1 alone had significantly lower adhesion scores than the controls.