H
Hongsheng Liu
Researcher at Boston University
Publications - 12
Citations - 1016
Hongsheng Liu is an academic researcher from Boston University. The author has contributed to research in topics: Medicine & Engineering. The author has an hindex of 7, co-authored 9 publications receiving 920 citations. Previous affiliations of Hongsheng Liu include Peking University.
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Journal ArticleDOI
Platelet-Rich Plasma: Growth Factors and Pro- and Anti-Inflammatory Properties
Hesham M. El-Sharkawy,Hesham M. El-Sharkawy,Alpdogan Kantarci,Jennifer Deady,Hatice Hasturk,Hongsheng Liu,Mohammad Alshahat,Thomas E. Van Dyke +7 more
TL;DR: PRP is a rich source of growth factors and promoted significant changes in monocyte-mediated proinflammatory cytokine/chemokine release and lipoxin A(4) (LXA(4)) generation, suggesting that PRP may suppress cytokine release, limit inflammation, and, thereby, promote tissue regeneration.
Journal ArticleDOI
Brd2 disruption in mice causes severe obesity without Type 2 diabetes.
Fangnian Wang,Hongsheng Liu,Wanda P. Blanton,Anna C. Belkina,Nathan K. LeBrasseur,Gerald V. Denis +5 more
TL;DR: Hypomorphic Brd2 shifts energy balance toward storage without causing glucose intolerance and may provide a novel model for obese metabolically healthy humans.
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Priming of neutrophil oxidative burst in diabetes requires preassembly of the NADPH oxidase.
Kazuhiro Omori,Taisuke Ohira,Yushi Uchida,Srinivas Ayilavarapu,Eraldo L. Batista,Motohiko Yagi,Tomoyuki Iwata,Hongsheng Liu,Hatice Hasturk,Alpdogan Kantarci,Thomas E. Van Dyke +10 more
TL;DR: Data suggest that HG and increased AGE prime neutrophils and increase oxidative stress inducing the translocation of p47phox to the cell membrane and preassembly with p22phox by stimulating a RAGE‐ERK1/2 pathway.
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Diabetes-Induced Oxidative Stress Is Mediated by Ca2+-Independent Phospholipase A2 in Neutrophils
Srinivas Ayilavarapu,Alpdogan Kantarci,Gabrielle Fredman,Oya Türkoğlu,Kazuhiro Omori,Hongsheng Liu,Tomoyuki Iwata,Motohiko Yagi,Hatice Hasturk,Thomas E. Van Dyke +9 more
TL;DR: Evidence is provided for the role of iPLA2 in enhanced superoxide generation in neutrophils from people with diabetes mellitus and presents an alternate pathway independent of protein kinase C and phosphatidic acid phosphohydrolase-1 hydrolase signaling.