H
Hongwei Si
Researcher at Tennessee State University
Publications - 34
Citations - 1904
Hongwei Si is an academic researcher from Tennessee State University. The author has contributed to research in topics: Genistein & Monocyte. The author has an hindex of 19, co-authored 28 publications receiving 1516 citations. Previous affiliations of Hongwei Si include Virginia Tech.
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Journal ArticleDOI
Combined Luteolin and Indole-3-Carbinol Synergistically Constrains ERα-Positive Breast Cancer by Dual Inhibiting Estrogen Receptor Alpha and Cyclin-Dependent Kinase 4/6 Pathway in Cultured Cells and Xenograft Mice.
Xiaoyong Wang,Xiaoyong Wang,Lijuan Zhang,Lijuan Zhang,Qi Dai,Hongzong Si,Longyun Zhang,Sakina E. Eltom,Hongwei Si +8 more
TL;DR: In this article, a combination of luteolin at 30 μM and indole-3-carbinol 40 μM (I3C40) was proposed to suppress ERα+ breast cancer cell proliferation.
Journal ArticleDOI
Correction: Aromokeye, R.; Si, H. Combined Curcumin and Luteolin Synergistically Inhibit Colon Cancer Associated with Notch1 and TGF-β Signaling Pathways in Cultured Cells and Xenograft Mice. Cancers 2022, 14, 3001
Rukayat Aromokeye,Hongwei Si +1 more
TL;DR: In the original publication as mentioned in this paper , the authors proposed a method to solve the problem of homonymity in the context of homology, which is called homology-based homology.
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How Does Ginsenoside Rh2 Mitigate Adipogenesis in Cultured Cells and Obese Mice
TL;DR: It is hypothesized that ginsenoside Rh2 attenuates adipogenesis through regulating the peroxisome proliferator-activated receptor gamma (PPAR-γ) pathway both in cells and obese mice and may be a potential agent in preventing obesity in vivo.
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The Aging of Adipocytes Increases Expression of Pro-Inflammatory Cytokines Chronologically.
Bulbul Ahmed,Hongwei Si +1 more
TL;DR: This paper showed that aging augmented chronic inflammation through the NF-κB signaling pathway in adipocytes and adipose tissue, and showed that the aging adipocytes chronologically accumulated lipids and increased pro-inflammatory markers interleukin-6 (IL-6), monocyte chemoattractant protein-1 (MCP-1), and tumor necrosis factor-alpha (TNF-α); at the same time, NFκB p50 markers were also increased while IκBα protein was decreased significantly in conditioned medium.
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Synergistic anti-inflammatory effects and mechanisms of the combination of resveratrol and curcumin in human vascular endothelial cells and rodent aorta.
TL;DR: In this article , a combination of curcumin and resveratrol was used to prevent TNF-α-induced vascular inflammation by suppressing NF-κB signaling in vitro and in vivo models.