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Hui Wang

Researcher at Capital Medical University

Publications -  54
Citations -  1287

Hui Wang is an academic researcher from Capital Medical University. The author has contributed to research in topics: Medicine & Internal medicine. The author has an hindex of 16, co-authored 37 publications receiving 1018 citations. Previous affiliations of Hui Wang include Harvard University & Sichuan University.

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Selective Anesthesia-induced Neuroinflammation in Developing Mouse Brain and Cognitive Impairment

TL;DR: The findings suggest the cellular basis and the potential prevention and treatment strategies for anesthesia-induced cognitive impairment, which may ultimately lead to safer anesthesia care and better postoperative outcomes for children.
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Anesthetics isoflurane and desflurane differently affect mitochondrial function, learning, and memory.

TL;DR: Inhalation anesthetic isoflurane has been shown to induce caspase activation and apoptosis, which are part of AD neuropathogenesis, through the mitochondria‐dependent apoptosis pathway, but the in vivo relevance, underlying mechanisms, and functional consequences remain largely to be determined.
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Age-dependent postoperative cognitive impairment and Alzheimer-related neuropathology in mice.

TL;DR: Data suggested that the peripheral surgery was able to induce cognitive impairment independent of general anesthesia, and that the combination of peripheral surgery with aging- or Alzheimer gene mutation-associated Aβ accumulation was needed for the POCD to occur.
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Cardiac involvement in COVID-19 patients: mid-term follow up by cardiovascular magnetic resonance.

TL;DR: In this paper, the authors evaluated mid-term cardiac sequelae in recovered COVID-19 patients by CMR and found that LGE-positive patients had significantly decreased LV peak global circumferential strain (GCS), RV peak GCS, RV peak global longitudinal strain, and peak GLS as compared to non-LGE patients.
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Role of distinct CD4(+) T helper subset in pathogenesis of oral lichen planus.

TL;DR: The roles of distinct CD4(+) Th subsets and their signature cytokines in determining disease severity and susceptibility of OLP are reviewed and the novel therapeutic strategies based on T lymphocytes subsets in OLP treatment are revealed.