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I-Shan Chen

Researcher at National Institutes of Natural Sciences, Japan

Publications -  15
Citations -  198

I-Shan Chen is an academic researcher from National Institutes of Natural Sciences, Japan. The author has contributed to research in topics: G protein-coupled inwardly-rectifying potassium channel & Chemistry. The author has an hindex of 7, co-authored 11 publications receiving 132 citations. Previous affiliations of I-Shan Chen include Wakayama Medical University & Graduate University for Advanced Studies.

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Ivermectin and its target molecules: shared and unique modulation mechanisms of ion channels and receptors by ivermectin.

TL;DR: An update and summary of recent progress in the identification of IVM targets, as well as their modulation mechanisms, through molecular structures, chimeras and site‐directed mutagenesis, and molecular docking and modelling studies is provided.
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Ivermectin activates GIRK channels in a PIP2-dependent, Gβγ-independent manner and an amino acid residue at the slide helix governs the activation

TL;DR: In this paper, the authors found that the G-protein-gated inwardly rectifying K+ (GIRK) channel is activated by IVM directly in a Gβγ-independent manner, but the activation is dependent on phosphatidylinositol-4,5-biphosphate (PIP2).
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A ciliary opsin in the brain of a marine annelid zooplankton is ultraviolet-sensitive, and the sensitivity is tuned by a single amino acid residue

TL;DR: It is demonstrated that the c-opsin can sense UV to activate intracellular signaling cascades and that it can directly bind exogenous all-trans-retinal in the brain of Platynereis dumerilii, suggesting that this c-opin regulates circadian signaling in a UV-dependent manner and thatIt does not require a supply of 11-cis-reteral for photoreception.
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Protein kinases modulate store-operated channels in pulmonary artery smooth muscle cells

TL;DR: SOC activity in PASMCs was stimulated by the inhibition of PKG and PKA and the activation of PKC, suggesting that the SOC could be a substrate of these protein kinases, which would regulate the intracellular concentration of calcium and pulmonary arteriopathy via SOC.