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Ian Ellwood
Researcher at University of California, San Francisco
Publications - 29
Citations - 1769
Ian Ellwood is an academic researcher from University of California, San Francisco. The author has contributed to research in topics: String field theory & Tachyon. The author has an hindex of 20, co-authored 28 publications receiving 1620 citations. Previous affiliations of Ian Ellwood include University of California, Berkeley & Lawrence Berkeley National Laboratory.
Papers
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Journal ArticleDOI
Thermoelectric Power of Single-Walled Carbon Nanotubes
James Hone,James Hone,Ian Ellwood,Ian Ellwood,M. Muno,M. Muno,Ari Mizel,Ari Mizel,Marvin L. Cohen,Marvin L. Cohen,Alex Zettl,Alex Zettl,Andrew G. Rinzler,Richard E. Smalley +13 more
TL;DR: In this paper, the temperature-dependent thermoelectric power (TEP) of crystalline ropes of single-walled carbon nanotubes was measured and it was shown that the TEP is large and hole-like at high temperatures and approaches zero as T{r_arrow}0.
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Synaptic activity unmasks dopamine D2 receptor modulation of a specific class of layer V pyramidal neurons in prefrontal cortex
TL;DR: D2Rs can elicit a Ca2+-channel-dependent afterdepolarization that powerfully modulates activity in specific prefrontal neurons through this mechanism, which might enhance outputs to subcortical structures, contribute to reward-related persistent firing, or increase the level of noise in prefrontal circuits.
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Proof of vanishing cohomology at the tachyon vacuum
Ian Ellwood,Martin Schnabl +1 more
TL;DR: In this paper, the authors proved Sen's third conjecture that there are no on-shell perturbative excitations of the tachyon vacuum in open bosonic string field theory.
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The Closed string tadpole in open string field theory
TL;DR: In this paper, a class of gauge invariant observables for marginal solutions and the tachyon vacuum were computed and shown to be related in a simple way to the closed-string tadpole on a disk with appropriate boundary conditions.
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Identifying specific prefrontal neurons that contribute to autism-associated abnormalities in physiology and social behavior.
Audrey C. Brumback,Ian Ellwood,Celia Kjaerby,Jillian Iafrati,Sarah Robinson,Anthony T. Lee,Tosha Patel,S. Nagaraj,F. Davatolhagh,Vikaas S. Sohal +9 more
TL;DR: It is suggested that multiple forms of autism may alter the physiology of specific deep-layer prefrontal neurons that project to subcortical targets, and a highly overlapping population—prefrontal D2R+ neurons—plays an important role in both normal and abnormal social behavior, such that targeting these cells can elicit potentially therapeutic effects.