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J

J. Conway

Researcher at University of Toronto

Publications -  11
Citations -  397

J. Conway is an academic researcher from University of Toronto. The author has contributed to research in topics: Transplantation & Canadian Cardiovascular Society. The author has an hindex of 6, co-authored 11 publications receiving 332 citations. Previous affiliations of J. Conway include University of Alberta.

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Presentation, diagnosis, and medical management of heart failure in children: Canadian Cardiovascular Society guidelines.

TL;DR: In this paper, the authors present guidelines for the recognition, diagnosis, and early medical management of pediatric heart failure in infancy, childhood, and adolescence, which are intended to assist practitioners in office-based or emergency room practice, who encounter children with undiagnosed heart disease and symptoms of possible HF.
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Delineating survival outcomes in children <10 kg bridged to transplant or recovery with the Berlin Heart EXCOR Ventricular Assist Device.

TL;DR: Overall results for children weighing <10 kg were inferior to those of their larger counterparts, primarily influenced by congenital heart disease and presence of elevated pre-implant bilirubin levels.
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Mortality and morbidity after retransplantation after primary heart transplant in childhood: an analysis from the registry of the International Society for Heart and Lung Transplantation.

TL;DR: Retransplantation after primary transplant in the pediatric age group, although feasible with similar early survival, is associated with decreased long-term survival and an increase in transplant-related morbidities.
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Enhanced Exercise Performance and Survival Associated With Evidence of Autonomic Reinnervation in Pediatric Heart Transplant Recipients

TL;DR: A majority of pediatric HTx recipients demonstrate evidence of reinnervation that is associated with functional outcomes, and studies to assess graft reinn conservation as a marker of long‐term prognosis are warranted.
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Development of donor-specific isohemagglutinins following pediatric ABO-incompatible heart transplantation.

TL;DR: The presence of DSI did increase the risk of cellular rejection but not antibody‐mediated rejection, allograft vasculopathy, graft loss or death, and their presence suggests that B‐cell tolerance is not the sole mechanism of graft acceptance.