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James A. Moses

Researcher at Stanford University

Publications -  91
Citations -  1750

James A. Moses is an academic researcher from Stanford University. The author has contributed to research in topics: Luria-Nebraska neuropsychological battery & Neuropsychology. The author has an hindex of 23, co-authored 90 publications receiving 1729 citations. Previous affiliations of James A. Moses include United States Department of Veterans Affairs & Ames Research Center.

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Cerebral Ventricular Size and Neuropsychological Impairment in Young Chronic Schizophrenics: Measurement by the Standardized Luria-Nebraska Neuropsychological Battery

TL;DR: Investigation of the relationship between size of cerebral ventricles in chronic schizophrenics and performance on a comprehensive neuropsychological battery found a multiple correlation between the ventricular brain ratio and Luria scores.
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Computed tomography in schizophrenics and normal volunteers. I. Fluid volume.

TL;DR: Results suggest that the findings and those based on measurements of planimetric ventricle-brain ratios (VBRs) are highly correlated, but that VBRs from one study may not be compared with those in another to establish population differences.
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A neuropsychological study of early onset schizophrenia

TL;DR: It is demonstrated that an early age of onset in schizophrenic illness is associated with impairment on tasks which involve motor and language abilities, functions linked to the frontal, temporal, and subcortical regions of the brain.
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Symptomatology and cognitive impairment associate independently with post-dexamethasone cortisol concentrations in unmedicated schizophrenic patients.

TL;DR: Serum cortisol concentrations were measured after dexamethasone administration in 21 neuroleptic-free schizophrenic inpatients and revealed a significant inverse correlation between a positive symptom grouping and both 8:00 AM and 4:00 PM PDC.
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Computed Tomography in Schizophrenics and Normal Volunteers: II. Cranial Asymmetry

TL;DR: No group differences were observed in cranial asymmetry in schizophrenics and normal volunteers, and methodologic flaws in earlier studies may account for this discrepancy.