James G. Traylor

TL;DR: The current literature on methamphetamine-induced changes in cardiovascular health is examined, the potential mechanisms regulating these varied effects are discussed, and deficiencies in understanding how to treat methamphetamine-associated cardiovascular dysfunction are highlighted.

TL;DR: It is concluded that both AlpA and AlpB contribute to H. pylori laminin binding, and identification of laminIn as the target of Alp a and AlPB will facilitate future investigations of host-pathogen interactions occurring during H.pylori infection.

TL;DR: The current data suggest that enhanced EphA2 signaling during endothelial cell activation perpetuates proinflammatory gene expression, which implicate E phA2 as a novel proinflammatory mediator and potential regulator of atherosclerotic plaque development.

TL;DR: It is demonstrated that in DSS colitis, Ang‐2 mediates inflammatory hemangiogenesis, lymphangiogenic and neutrophil infiltration to reduce some, but not all clinical features of IBD.

TL;DR: Interleukin-19 is a potent inhibitor of experimental atherosclerosis, with diverse mechanisms including immune cell polarization, decrease in macrophage adhesion, and decrease in gene expression that may identify IL-19 as a novel therapeutic to limit vascular inflammation.