Showing papers by "James L. Weber published in 1988"

TL;DR: The mechanisms responsible for the protective immunity appeared to be cellular effector mechanisms and it appeared that cellular effectors mechanisms are probably involved.

TL;DR: Sequences from the Camp and FCR3 strains are very close and are also nearly identical to the partial cDNA sequence of the acidic basic repeated antigen (ABRA) protein from the FC27 strain, a candidate component of a malaria vaccine.

TL;DR: An interspersed repetitive DNA element from the Camp strain of the human malaria parasite Plasmodium falciparum was cloned and sequenced, indicating that the repeat family represented by this element could be transposable.

TL;DR: Using pulsed field gel electrophoresis, eight chromosomal DNA molecules from clone 7G8 of the Plasmodium falciparum Brazilian isolate IM TM22 were resolved and the restriction enzyme NotI appeared to produce only one cut in the entire IMTM22 genome.