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Janet E Foley

Researcher at University of California, Davis

Publications -  301
Citations -  9396

Janet E Foley is an academic researcher from University of California, Davis. The author has contributed to research in topics: Anaplasma phagocytophilum & Tick. The author has an hindex of 47, co-authored 283 publications receiving 8330 citations. Previous affiliations of Janet E Foley include University of California & University of Liverpool.

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Feline Infectious Peritonitis Viruses Arise by Mutation from Endemic Feline Enteric Coronaviruses

TL;DR: It was concluded that FIPVs evolved as mutants of FECVs, and the order of descent is from FECV to FIPV.
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Causal inference in disease ecology: investigating ecological drivers of disease emergence

TL;DR: Some of the barriers to advancing the understanding of causation in disease ecology are outlined and some solutions for investigating large-scale ecological drivers, such as global warming, pollution, and land-use change are offered.
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Urban habituation, ecological connectivity and epidemic dampening: the emergence of Hendra virus from flying foxes (Pteropus spp.)

TL;DR: This work examines the transmission dynamics of an emerging zoonotic paramyxovirus, Hendra virus (HeV), in its endemic host, Australian Pteropus bats, and provides the first detailed mechanistic framework for understanding the sporadic temporal pattern of HeV emergence, and of the urban/peri-urban distribution of HeVs outbreaks in horses and people.
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Reproduction and nutritional stress are risk factors for Hendra virus infection in little red flying foxes (Pteropus scapulatus)

TL;DR: Data showed that pregnant and lactating females had significantly higher risk of infection, which may explain previously observed temporal associations between HeV outbreaks and flying fox birthing periods and causal links between anthropogenic environmental change and HeV emergence are suggested.
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Amplification of Ehrlichial DNA from Dogs 34 Months after Infection with Ehrlichia canis

TL;DR: It was concluded that PCR of DNA extracted from splenic aspirates is a reliable method for determining the carrier state of CME and suggests that the spleen is the organ most likely to harbor E. canis parasites during the subclinical phase and the last organ to accommodate the parasite before elimination.