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Janet E. Price

Researcher at University of Texas MD Anderson Cancer Center

Publications -  122
Citations -  8550

Janet E. Price is an academic researcher from University of Texas MD Anderson Cancer Center. The author has contributed to research in topics: Metastasis & Cancer. The author has an hindex of 48, co-authored 122 publications receiving 8221 citations. Previous affiliations of Janet E. Price include University of Texas at Austin & University of Texas Health Science Center at Houston.

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Journal ArticleDOI

Amplification of telomeric DNA directly correlates with metastatic potential of human and murine cancers of various histological origin.

TL;DR: Telomeres, repeated DNA sequences that guard the ends of chromosomes, serve as a checkpoint for cell-cycle progression and regulate cell senescence and apoptosis, and amplification of telomeres is directly correlated with invasive and metastatic potential of murine or human tumor cells.
Book ChapterDOI

Spontaneous and experimental metastasis models: nude mice.

TL;DR: The appropriate design of animal models using nude mice, and established human tumor cell lines, assists in the generation of novel information about the metastatic phenotype, and provides a valuable, preclinical system for testing anti-metastatic therapies.

Curcumin Suppresses the Paclitaxel-Induced Nuclear Factor-KB Pathway in Breast Cancer Cells and Inhibits LungMetastasis of Human Breast Cancer in NudeMice

TL;DR: Curcumin has been shown to suppress NFnB activation induced by various inflammatory stimuli (6) through inhibition of the activation of InBa kinase (IKK) activity as discussed by the authors.
Journal ArticleDOI

Antiproliferative activity of liposome-encapsulated transforming growth factor-beta against MDA-MB-435 human breast carcinoma cells.

TL;DR: The lipophilic nature of TGF-beta, which resulted in a high capture ratio in liposome, coupled with exceptional stability, suggested that liposomes could be a carrier for the in vivo use of T GF-beta.
Journal ArticleDOI

Effects of altering surface glycoprotein composition on metastatic colonisation potential of murine mammary tumour cells.

TL;DR: It is concluded that a clear answer to whether surface glycoprotein composition has a decisive role in pulmonary colonisation by these mammary tumour cells requires introduction of stable heritable traits into tumour cell populations by genetic manipulation.