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Jason D. Lee

Researcher at Princeton University

Publications -  208
Citations -  13130

Jason D. Lee is an academic researcher from Princeton University. The author has contributed to research in topics: Gradient descent & Artificial neural network. The author has an hindex of 56, co-authored 196 publications receiving 10472 citations. Previous affiliations of Jason D. Lee include Stanford University & Kaiser Permanente.

Papers
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Proceedings Article

Gradient descent finds global minima of deep neural networks

TL;DR: This paper showed that gradient descent achieves zero training loss in polynomial time for a deep over-parameterized neural network with residual connections (ResNet) and further extended their analysis to deep residual convolutional neural networks and obtained a similar convergence result.
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Exact post-selection inference, with application to the lasso

TL;DR: A general approach to valid inference after model selection by the lasso is developed to form valid confidence intervals for the selected coefficients and test whether all relevant variables have been included in the model.
Proceedings Article

Gradient Descent Only Converges to Minimizers

TL;DR: The authors showed that gradient descent converges to a local minimizer almost surely with random initialization by applying the Stable Manifold Theorem from dynamical systems theory, which is proved by applying stable manifold theorem to gradient descent.
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Theoretical Insights Into the Optimization Landscape of Over-Parameterized Shallow Neural Networks

TL;DR: In this paper, the problem of learning a shallow neural network that best fits a training data set was studied in the over-parameterized regime, where the numbers of observations are fewer than the number of parameters in the model.
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Ubiquitination-dependent mechanisms regulate synaptic growth and function

TL;DR: It is shown that ubiquitin-dependent mechanisms regulate synaptic development at the Drosophila neuromuscular junction (NMJ) and genetic interactions between fat facets and highwire suggest that synaptic development may be controlled by the balance between positive and negative regulators of ubiquitination.