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Jean-Jacques Risso

Researcher at Aix-Marseille University

Publications -  74
Citations -  1567

Jean-Jacques Risso is an academic researcher from Aix-Marseille University. The author has contributed to research in topics: Decompression sickness & Tissue plasminogen activator. The author has an hindex of 19, co-authored 72 publications receiving 1423 citations. Previous affiliations of Jean-Jacques Risso include University of the Mediterranean.

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Differential vesicular distribution and trafficking of MMP-2, MMP-9, and their inhibitors in astrocytes.

TL;DR: Investigation in cultured reactive astrocytes of the intracellular distribution, transport, and secretion of M MP‐2, MMP‐9, TIMP‐1, and TIMP-2 suggests that these proteinases and their inhibitors use different pathways for trafficking and secretion for distinct astroCytic functions.
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Neuroprotective effects of xenon: a therapeutic window of opportunity in rats subjected to transient cerebral ischemia

TL;DR: It is shown that xenon, administered at subanesthetic doses, offers global neuroprotection from reduction of neurotransmitter release induced by ischemia, a critical event known to be involved in excitotoxicity, to reduction of subsequent cell injury and neuronal death.
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Gamma-aminobutyric acid neuropharmacological investigations on narcosis produced by nitrogen, argon, or nitrous oxide.

TL;DR: The results support a selective antagonism by gabazine and flumazenil of the narcotic action of nitrogen and argon.
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Vesicular trafficking and secretion of matrix metalloproteinases-2, -9 and tissue inhibitor of metalloproteinases-1 in neuronal cells

TL;DR: In this paper, the gelatinases MMP-2 and TIMP-1 fused to GFP were expressed in N2a neuroblastoma and primary neuronal cells to study trafficking and secretion using real-time video-microscopy, imaging, electron microscopy and biochemical approaches.
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Vitamin D2 Potentiates Axon Regeneration

TL;DR: Data indicate that vitamin D potentiates axon regeneration and functional recovery was not impaired by vitamin D supplementation, and pharmacological studies with various concentrations of the two forms of vitamin D are now required before recommending this molecule as a potential supplemental therapeutic approach following nerve injury.