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Jeroen Nauta

Researcher at Erasmus University Rotterdam

Publications -  72
Citations -  2786

Jeroen Nauta is an academic researcher from Erasmus University Rotterdam. The author has contributed to research in topics: Transplantation & Renal function. The author has an hindex of 29, co-authored 72 publications receiving 2588 citations. Previous affiliations of Jeroen Nauta include Boston Children's Hospital & University of Washington.

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Microalbuminuria and Lower Glomerular Filtration Rate at Young Adult Age in Subjects Born Very Premature and after Intrauterine Growth Retardation

TL;DR: The data suggest that intrauterine growth-retarded subjects born very premature have an increased risk to develop progressive renal failure in later life, and IUGR is associated with unfavorable renal functions at young adult age in subjects Born very premature.
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Role of truncating mutations in MME gene in fetomaternal alloimmunisation and antenatal glomerulopathies

TL;DR: It is shown that disease caused by fetomaternal alloimmunisation secondary to a genetic defect is not restricted to blood cells and should be considered as a leading cause of membranous glomerulopathy early in life.
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Deregulation of cell survival in cystic and dysplastic renal development.

TL;DR: It is speculated that deregulation of cell survival in these kidneys may reflect incomplete tissue maturation, and may contribute to the progressive destruction of functional kidney tissue in polycystic kidneys and the spontaneous involution reported in cystic dysplastic kidneys.
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Is blood pressure increased 19 years after intrauterine growth restriction and preterm birth? A prospective follow-up study in the Netherlands

TL;DR: The prevalence of hypertension is high in individuals who were born preterm when compared with the general population and no support to the hypothesis that low birth weight is associated with increased BP at young adult age can be given.
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Renal and biliary abnormalities in a new murine model of autosomal recessive polycystic kidney disease.

TL;DR: The hyperplastic abnormalities in both renal and biliary epithelium make this new mouse strain a good model for the study of the dual organ cellular pathophysiology of ARPKD.