J
Jincai Luo
Researcher at Peking University
Publications - 32
Citations - 1860
Jincai Luo is an academic researcher from Peking University. The author has contributed to research in topics: Angiogenesis & Vascular endothelial growth factor. The author has an hindex of 18, co-authored 29 publications receiving 1574 citations. Previous affiliations of Jincai Luo include University of Tokyo & Beth Israel Deaconess Medical Center.
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Journal ArticleDOI
Shp2 Regulates Src Family Kinase Activity and Ras/Erk Activation by Controlling Csk Recruitment
Si Qing Zhang,Wentian Yang,Maria I. Kontaridis,Trever G. Bivona,Gengyun Wen,Toshiyuki Araki,Jincai Luo,Julie A. Thompson,Burkhart Schraven,Mark R. Philips,Benjamin G. Neel +10 more
TL;DR: It is shown that Shp2 promotes Src family kinase (SFK) activation by regulating the phosphorylation of the Csk regulator PAG/Cbp, thereby controlling Csk access to SFKs.
Journal ArticleDOI
Meningeal lymphatic vessels regulate brain tumor drainage and immunity.
Xueting Hu,Qiuping Deng,Lu Ma,Qingqing Li,Yidong Chen,Yuhan Liao,Fan Zhou,Chen Zhang,Linlin Shao,Jun Feng,Tubao He,Weihai Ning,Yan Kong,Yingqing Huo,Aibin He,Aibin He,Bing Liu,Jingjing Zhang,Ralf H. Adams,Yulong He,Fuchou Tang,Fuchou Tang,Xiu-Wu Bian,Jincai Luo +23 more
TL;DR: The results of this study demonstrate the functional aspects of MLVs as classic lymphatic vasculature, but also highlight that they are essential in generating an efficient immune response against brain tumors.
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Involvement of COX-2 in VEGF-induced angiogenesis via P38 and JNK pathways in vascular endothelial cells
TL;DR: COX-2 plays an important role in VEGF-induced angiogenesis via p38 and JNK kinase activation pathways, and the findings suggest that the cardioprotective role of COx-2 may be, at least in part, through its angiogenic activity.
Journal Article
Significant Expression of Vascular Endothelial Growth Factor/Vascular Permeability Factor in Mouse Ascites Tumors
TL;DR: A complicated relationship may exist between the VEGF production and angiogenesis associated with ascites tumor in vivo, and its importance may vary according to tumor origin.
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VEGFR-3 ligand-binding and kinase activity are required for lymphangiogenesis but not for angiogenesis
TL;DR: It is shown that signaling mediated via VEGFR-3 activation by its cognate ligands (VEGF-C/-D) is not required for angiogenesis, and that VEGfr-3 may play a role in this process by modulating V EGFR-2-mediated signals.