J
Jing Shi
Researcher at Huazhong University of Science and Technology
Publications - 71
Citations - 3395
Jing Shi is an academic researcher from Huazhong University of Science and Technology. The author has contributed to research in topics: Apoptosis & Ischemia. The author has an hindex of 24, co-authored 67 publications receiving 2475 citations. Previous affiliations of Jing Shi include Chinese Ministry of Health & Chinese Ministry of Education.
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Journal ArticleDOI
Risk factors for severity and mortality in adult COVID-19 inpatients in Wuhan.
Xiaochen Li,Xiaochen Li,Shuyun Xu,Shuyun Xu,Muqing Yu,Muqing Yu,Ke Wang,Ke Wang,Yu Tao,Yu Tao,Ying Zhou,Ying Zhou,Jing Shi,Jing Shi,Min Zhou,Min Zhou,Bo Wu,Zhenyu Yang,Zhenyu Yang,Cong Zhang,Cong Zhang,Junqing Yue,Junqing Yue,Zhiguo Zhang,Harald Renz,Xiansheng Liu,Xiansheng Liu,Jungang Xie,Jungang Xie,Min Xie,Min Xie,Jianping Zhao,Jianping Zhao +32 more
TL;DR: Survival analysis revealed that male, elder age, leukocytosis, high LDH level, cardiac injury, hyperglycemia, and high-dose corticosteroid use were associated with death in patients with severe COVID-19.
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Gendered effects on inflammation reaction and outcome of COVID-19 patients in Wuhan.
Lu Qin,Lu Qin,Xiaochen Li,Xiaochen Li,Jing Shi,Jing Shi,Muqing Yu,Muqing Yu,Ke Wang,Ke Wang,Yu Tao,Yu Tao,Ying Zhou,Ying Zhou,Min Zhou,Min Zhou,Shuyun Xu,Shuyun Xu,Bo Wu,Zhenyu Yang,Zhenyu Yang,Cong Zhang,Cong Zhang,Junqing Yue,Junqing Yue,Chongsheng Cheng,Chongsheng Cheng,Xiansheng Liu,Xiansheng Liu,Min Xie,Min Xie +30 more
TL;DR: The gendered differences in inflammation reaction, and the association with severity and mortality of COVID‐19 are studied to study the underlying mechanism of sex predisposition.
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Activation of NLRP3 inflammasome in peripheral nerve contributes to paclitaxel-induced neuropathic pain
Min Jia,Cai-Hua Wu,Fang Gao,Hong-Chun Xiang,Ning Sun,Ping Peng,Jing-Jing Li,Xiao-Cui Yuan,Hong-Ping Li,Xian-Fang Meng,Bo Tian,Jing Shi,Man Li +12 more
TL;DR: The administration of a non-specific reactive oxygen species scavenger, phenyl-N-tert-butylnitrone, markedly alleviated mechanical allodynia and inhibited the activation of NLRP3 inflammasome in infiltrated macrophages of L4-6 dorsal root ganglia and sciatic nerve of the paclitaxel-induced neuropathic pain model.
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Cdk5-Dependent Activation of Neuronal Inflammasomes in Parkinson's Disease.
Pei Zhang,Xiao-Yun Shao,Xiao-Yun Shao,Guangjian Qi,Guangjian Qi,Qiang Chen,Qiang Chen,Lu-Lu Bu,Lu-Lu Bu,Li-Jun Chen,Li-Jun Chen,Jing Shi,Jing Shi,Jie Ming,Bo Tian,Bo Tian +15 more
TL;DR: Inflammasomes, which mediate the activation of caspase‐1 and maturation of IL‐1β and IL‐18, have been unambiguously verified to participate in many diseases, such as lung diseases, infectious diseases and Alzheimer's disease, but the relation between Parkinson's disease and inflammasome is poorly understood.
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Endogenous Anandamide and Cannabinoid Receptor-2 Contribute to Electroacupuncture Analgesia in Rats
TL;DR: Electroacupuncture increases the anandamide level in inflammatory skin tissues, and CB2 receptors contribute to the analgesic effect of electroacupuncture in a rat model of inflammatory pain, which suggests that EA potentiates the local release of endogenousAnandamide from inflamed tissues.