J
Jing-Ting Mai
Researcher at Sun Yat-sen University
Publications - 19
Citations - 431
Jing-Ting Mai is an academic researcher from Sun Yat-sen University. The author has contributed to research in topics: Heart failure & Inflammation. The author has an hindex of 10, co-authored 18 publications receiving 285 citations.
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Journal ArticleDOI
Clinical effectiveness of telemedicine for chronic heart failure: a systematic review and meta-analysis
TL;DR: Overall, telemedicine was shown to be beneficial, with home-based teletransmission effectively reducing all-cause mortality and HF-related hospital admission, length of stay and mortality in patients with HF.
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Hydrogen sulfide suppresses endoplasmic reticulum stress-induced endothelial-to-mesenchymal transition through Src pathway
Ru Ying,Xiao-Qiao Wang,Ying Yang,Zhen-Jie Gu,Jing-Ting Mai,Qiong Qiu,Yangxin Chen,Jingfeng Wang +7 more
TL;DR: H2S could protect against ER stress-induced EndMT through Src pathway, which may be a novel role for the cardioprotection of H2S.
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Altered serum levels of IL-33 in patients with advanced systolic chronic heart failure: correlation with oxidative stress.
Hai-Feng Zhang,Shuang-Lun Xie,Yangxin Chen,Jing-Ting Mai,Jingfeng Wang,Wa-Li Zhu,Li-Guang Zhu +6 more
TL;DR: In advanced CHF, IL-33 may exert anti-oxidation effects, which may be overwhelmed by concurrently elevated levels of sST2, which is positively correlated with markers of CHF severity.
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Macrophage-derived foam cells impair endothelial barrier function by inducing endothelial-mesenchymal transition via CCL-4.
Ying Yang,Nian-sang Luo,Ru Ying,Yong Xie,Jia-Yuan Chen,Xiao-Qiao Wang,Zhen-Jie Gu,Jing-Ting Mai,Liu Wenhao,Mao-Xiong Wu,Zhi-Teng Chen,Yong-Biao Fang,Hai-Feng Zhang,Zhiyi Zuo,Jingfeng Wang,Yangxin Chen +15 more
TL;DR: The data of the present study suggest that M1-FCs induce EndMT by upregulating CCL-4, and increase endothelial permeability and monocyte adhesion, which may help to elucidate the important role of EndMT in the development of atherosclerosis.
Journal ArticleDOI
LncRNA H19 governs mitophagy and restores mitochondrial respiration in the heart through Pink1/Parkin signaling during obesity.
Shaohua Wang,Xiao-Lin Zhu,Fei Wang,Si-Xu Chen,Zhi-Teng Chen,Qiong Qiu,Liu Wenhao,Mao-Xiong Wu,Bingqing Deng,Yong Xie,Jing-Ting Mai,Ying Yang,Jingfeng Wang,Hai-Feng Zhang,Yangxin Chen +14 more
TL;DR: In this paper, the effects of H19 on metabolic disorders, mitochondrial respiratory function, and mitophagy were investigated, and the regulatory mechanisms of palmitic acid (PA)-treated H9c2 cell and Lep−/− mice were used to investigate cardiac metabolic disorders in vitro and in vivo.