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Jinyoung Shin

Researcher at Konkuk University

Publications -  102
Citations -  1653

Jinyoung Shin is an academic researcher from Konkuk University. The author has contributed to research in topics: National Health and Nutrition Examination Survey & Odds ratio. The author has an hindex of 19, co-authored 100 publications receiving 1243 citations. Previous affiliations of Jinyoung Shin include Samsung & Yonsei University.

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Mesenchymal stem cells enhance autophagy and increase β-amyloid clearance in Alzheimer disease models

TL;DR: It is suggested that mesenchymal stem cells significantly enhance autolysosome formation and clearance of Aβ in AD models, which may lead to increased neuronal survival against Aβ toxicity.
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Increased arterial stiffness in healthy subjects with high-normal glucose levels and in subjects with pre-diabetes

TL;DR: FPG has an independent, positive association with ba-PWV in non-diabetic subjects after correcting for confounding variables, including age, sex, BMI, blood pressure, resting heart rate, hs-CRP, lipid profile, and behavioral habits.
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Long-term exposure to ambient air pollutants and mental health status: A nationwide population-based cross-sectional study.

TL;DR: Air pollution exposure was positively associated with high concentrations of PM10, NO2, and CO after adjusting for confounding factors and men were at increased risk of stress, poor QoL, and depressiveness from air pollution exposure than were women.
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Effect of fruits and vegetables on metabolic syndrome: a systematic review and meta-analysis of randomized controlled trials

TL;DR: The results suggest an inverse association between fruit and vegetable consumption and diastolic blood pressure in metabolic syndrome patients, and no statistically significant differences found according to the intervention period and provision type.
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Mesenchymal Stem Cells Inhibit Transmission of α-Synuclein by Modulating Clathrin-Mediated Endocytosis in a Parkinsonian Model

TL;DR: The present data indicated that MSCs exert neuroprotective properties through inhibition of extracellular α-synuclein transmission, suggesting that the property of M SCs may act as a disease-modifying therapy in subjects with α- synucleinopathies.