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Jodi F. Hedges

Researcher at Montana State University

Publications -  39
Citations -  1175

Jodi F. Hedges is an academic researcher from Montana State University. The author has contributed to research in topics: T cell & Innate immune system. The author has an hindex of 18, co-authored 39 publications receiving 969 citations.

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Intrinsic Signal Amplification by Type-III CRISPR-Cas Systems Provides a Sequence-Specific Viral Diagnostic

TL;DR: The type III CRISPR-Cas system is repurposed for sensitive and sequence specific detection of SARS-CoV-2 in an assay that can be performed in one hour or less and shows that amplified products of the Cas10-polymerase are detectable using colorimetric or fluorometric readouts.
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Oral delivery of oligomeric procyanidins in Apple Poly® enhances type I IFN responses in vivo

TL;DR: It is confirmed that OPCs from two different sources significantly increased pSTAT1, whereas a monomeric form of procyanidin did not, and this suggests a unique, nonantioxidant aspect of O PCs that is broadly applicable to many disease settings.
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Adjuvant materials that enhance bovine γδ T cell responses.

TL;DR: This review focuses on recent results obtained using two adjuvant therapies for cattle that can be used to help mitigate infection using bovine γδ T cells, which are important lymphocytes of the innate immune system and of particular importance to ruminant immunological health.
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Titers, Prevalence, and Duration of SARS-CoV-2 Antibodies in a Local COVID-19 Outbreak and Following Vaccination

TL;DR: It was found that antibody responses induced by vaccination were significantly higher than those induced by natural infection, suggesting that vaccination is still critical even for those naturally infected or diagnosed with COVID-19.
Posted ContentDOI

SARS-CoV-2 genomic surveillance identifies naturally occurring truncations of ORF7a that limit immune suppression

TL;DR: In this article, the C-terminal truncation of ORF7a has been shown to result in distinct changes in interferon stimulated gene expression, and these changes affect viral mechanisms responsible for suppressing the immune response.